Katheleen Gardiner

University of Colorado Anschutz Medical Campus — United States

Medicine · Health Sciences

43h-index10.8kcitations202works0.02yr avg
OpenAlex↗Google Scholar↗Lab page↗

Accepting Students?

No reports yet. Know if this professor is taking students?

Research Topics

Down syndrome and intellectual disability research(49), Chromosomal and Genetic Variations(30), Genetics and Neurodevelopmental Disorders(30), RNA and protein synthesis mechanisms(29), Genomics and Phylogenetic Studies(27)

Publications202 total

The innate immune system stimulating cytokine GM-CSF improves learning/memory and interneuron and astrocyte brain pathology in Dp16 Down syndrome mice and improves learning/memory in wild-type mice
Neurobiology of Disease·2022· 25 citedOpen Access
Trisomy of Human Chromosome 21 Orthologs Mapping to Mouse Chromosome 10 Cause Age and Sex-Specific Learning Differences: Relevance to Down Syndrome
Genes·2021· 3 citedOpen Access
Context Fear Conditioning in Down Syndrome Mouse Models: Effects of Trisomic Gene Content, Age, Sex and Genetic Background
Genes·2021· 6 citedOpen Access
Pro-inflammatory Cytokine GM-CSF Improves Learning/Memory and Brain Pathology in Dp16 Down Syndrome Mice and Improves Learning/Memory in Wild-Type Mice
2021· 5 citedOpen Access
Altered Protein Profiles During Epileptogenesis in the Pilocarpine Mouse Model of Temporal Lobe Epilepsy
Frontiers in Neurology·2021· 18 citedOpen Access
All Creatures Great and Small: New Approaches for Understanding Down Syndrome Genetics
Trends in Genetics·2020· 43 citedOpen Access
P4‐032: GM‐CSF REVERSES MEMORY DEFICITS IN NORMAL AGED MICE AND IN THE DP(16)1YEY MOUSE MODEL OF DOWN SYNDROME
Alzheimer s & Dementia·2019Open Access
Nrf2 stabilization prevents critical oxidative damage in Down syndrome cells
Aging Cell·2018· 58 citedOpen Access
P2‐162: GM‐CSF REVERSES MEMORY DEFICITS IN THE DP16 MOUSE MODEL OF DOWN SYNDROME
Alzheimer s & Dementia·2018· 1 cited
RPPAware: A software suite to preprocess, analyze and visualize reverse phase protein array data
Journal of Bioinformatics and Computational Biology·2018· 1 cited
The GABA A α5-selective Modulator, RO4938581, Rescues Protein Anomalies in the Ts65Dn Mouse Model of Down Syndrome
Neuroscience·2017· 10 cited
Age exacerbates abnormal protein expression in a mouse model of Down syndrome
Neurobiology of Aging·2017· 19 cited
Issue Information
Prenatal Diagnosis·2017· 1 citedOpen Access
Autismo y síndrome de Down: ajustar el diagnóstico y profundizar en la genética
Dialnet (Universidad de la Rioja)·2017
Erratum: Down Syndrome: Genes, Model Systems, and Progress towards Pharmacotherapies and Clinical Trials for Cognitive Deficits
Figshare·2017Open Access
The co‐occurrence of Down syndrome and autism spectrum disorder: is it because of additional genetic variations?
Prenatal Diagnosis·2016· 21 cited
Mouse models of Down syndrome: gene content and consequences
Mammalian Genome·2016· 172 citedOpen Access
The establishment of DOHaD working groups in Australia and New Zealand
Journal of Developmental Origins of Health and Disease·2016· 9 cited
Changing Paradigms in Down Syndrome: The First International Conference of the Trisomy 21 Research Society
Molecular Syndromology·2016· 21 citedOpen Access
Sex differences in protein expression in the mouse brain and their perturbations in a model of Down syndrome
Biology of Sex Differences·2015· 45 citedOpen Access
page 1 of 11Next →

Frequent Co-authors

Muriel T. Davisson(5), Norman R. Pace(4), A. Ranjitha Dhanasekaran(4), Alberto C. S. Costa(4), Md. Mahiuddin Ahmed(4), Terence L. Marsh(3), Stylianos E. Antonarakis(3), Roger H. Reeves(3), David Patterson(3), Georges Lutfalla(3), Xiaolu Sturgeon(3), Dobromir Slavov(3), David Patterson(2), Ira T. Lott(2), Jérôme Reboul(2), Lawrence Bechtel(2), Ellen C. Akeson(2), Gilles Uzé(2), David A. Patterson(2), Brahim Aı̈ssani(2)