Jonathan C. Makielski

University of Wisconsin–Madison — United States

Medicine · Health Sciences

53h-index10.3kcitations183works0.02yr avg
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Research Topics

Cardiac electrophysiology and arrhythmias(126), Ion channel regulation and function(117), Cardiac Ischemia and Reperfusion(29), Receptor Mechanisms and Signaling(22), Neuroscience and Neural Engineering(14)

Publications181 total

Caveolin-3 and Caveolae regulate ventricular repolarization
Journal of Molecular and Cellular Cardiology·2023· 10 citedOpen Access
Electrocardiographic and Echocardiographic Features of Carnitine-Deficient Animal Model
Journal of Biomedical Nanotechnology·2022
Does Anatomic Phenotype of Mitral Annular Disjunction Impact Survival? An Autopsy-Based Retrospective Study
Journal of Cardiovascular Development and Disease·2021· 2 citedOpen Access
Association of Mitral Annular Disjunction With Premature Cardiac Mortality in a Large Series of Autopsies
Journal of the American College of Cardiology·2021· 13 cited
Expression defect of the rare variant/Brugada mutation R1512W depends upon the SCN5A splice variant background and can be rescued by mexiletine and the common polymorphism H558R
Channels·2021· 3 citedOpen Access
Aligned human cardiac syncytium for in vitro analysis of electrical, structural, and mechanical readouts
Biotechnology and Bioengineering·2020· 20 citedOpen Access
Genetic Loss of <i>I</i> <sub>K1</sub> Causes Adrenergic-Induced Phase 3 Early Afterdepolariz ations and Polymorphic and Bidirectional Ventricular Tachycardia
Circulation Arrhythmia and Electrophysiology·2020· 23 citedOpen Access
A Singular Signature
Circulation·2019Open Access
Deletion of Sulfonylurea Receptor 2 in the Adult Myocardium Enhances Cardiac Glucose Uptake and Is Cardioprotective
JACC Basic to Translational Science·2019· 8 citedOpen Access
Abstract 427: Inhibition of Myocardial Romk Channels Blocks Ischemic Preconditioning Induced Cardio-protection
Circulation Research·2018
Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function
Heliyon·2018· 6 citedOpen Access
Mexiletine rescues a mixed biophysical phenotype of the cardiac sodium channel arising from the SCN5A mutation, N406K, found in LQT3 patients
Channels·2018· 17 citedOpen Access
Abstract 16578: Two Cysteine S-nitrosylation Sites on the Cardiac Sodium Channel Are Responsible for Increased Late Sodium Current in Long QT Syndromes Type 9 & Type 12 but Not Type 3
Circulation·2017
Sudden Infant Death Syndrome
Elsevier eBooks·2017
An East Asian Common Variant Vinculin P.Asp841His Was Associated With Sudden Unexplained Nocturnal Death Syndrome in the Chinese Han Population
Journal of the American Heart Association·2017· 11 citedOpen Access
Vinculin variant M94I identified in sudden unexplained nocturnal death syndrome decreases cardiac sodium current
Scientific Reports·2017· 5 citedOpen Access
Molecular pathological study on LRRC10 in sudden unexplained nocturnal death syndrome in the Chinese Han population
International Journal of Legal Medicine·2016· 13 citedOpen Access
Abstract 16628: Perturbations in IK1 and Calcium Handling Underlie CPVT-like Phenotype in a Cardiac-specific R67Q-KCNJ2 Mutant Mouse
Circulation·2016
Complexity of AF genetic susceptibility begets complexity of interpretation
Heart Rhythm·2016
Does Sudden Unexplained Nocturnal Death Syndrome Remain the Autopsy-Negative Disorder: A Gross, Microscopic, and Molecular Autopsy Investigation in Southern China
Mayo Clinic Proceedings·2016· 33 citedOpen Access
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Frequent Co-authors

Michael J. Ackerman(25), David J. Tester(18), Carmen R. Valdivia(16), Craig T. January(10), Bin Ye(8), Zheng Fan(7), Argelia Medeiros‐Domingo(7), Jielin Pu(5), Gianrico Farrugia(5), Bi-Hua Tan(5), Timothy J. Kamp(4), Kazuo Ueda(4), William A. Chutkow(4), Charles Burant(4), Elizabeth M. McNally(4), Peter R. Strege(4), John W. Kyle(4), Matteo Vatta(3), Lee L. Eckhardt(3), Stacie Kroboth(3)