Michael Hernandez

World Wide Web Consortium — United States

Medicine · Health Sciences

12h-index1.2kcitations26works36.82yr avg
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Research Topics

Alzheimer's disease research and treatments(11), Neuroinflammation and Neurodegeneration Mechanisms(11), S100 Proteins and Annexins(5), Long-Term Effects of COVID-19(4), Protein Structure and Dynamics(3)

Publications21 total

Prevention of C5aR1 signaling delays microglial inflammatory polarization, favors clearance pathways and suppresses cognitive loss
Molecular Neurodegeneration·2017· 104 citedOpen Access
Cell-specific deletion of C1qa identifies microglia as the dominant source of C1q in mouse brain
Journal of Neuroinflammation·2017· 344 citedOpen Access
C5a Increases the Injury to Primary Neurons Elicited by Fibrillar Amyloid Beta
ASN NEURO·2017· 47 citedOpen Access
Contribution of C5aR1 to Neuroinflammation and Neuronal Dysfunction in Alzheimer’s Disease
eScholarship (California Digital Library)·2017Open Access
Additional file 7: of Prevention of C5aR1 signaling delays microglial inflammatory polarization, favors clearance pathways and suppresses cognitive loss
Figshare·2017Open Access
Additional file 8: of Prevention of C5aR1 signaling delays microglial inflammatory polarization, favors clearance pathways and suppresses cognitive loss
Figshare·2017Open Access
Additional file 6: of Prevention of C5aR1 signaling delays microglial inflammatory polarization, favors clearance pathways and suppresses cognitive loss
Figshare·2017Open Access
Role of microglial C5aR1 in the Arctic Alzheimer's Disease mouse model
Immunobiology·2016
Microglia specific ablation of C1q gene definitively demonstrates microglia as the dominant source of C1q in both normal mouse brain and in models of Alzheimer's disease
Immunobiology·2016
Silencing of Tumor Necrosis Factor Receptor-1 in Human Lung Microvascular Endothelial Cells Using Particle Platforms for siRNA Delivery
Current Drug Targets·2015· 3 cited
Elimination of Microglia Improves Functional Outcomes Following Extensive Neuronal Loss in the Hippocampus
Journal of Neuroscience·2015· 268 citedOpen Access
A conformation-switching fluorescent protein probe for detection of alpha synuclein oligomers
Chemical Communications·2013· 15 cited
C1q-induced LRP1B and GPR6 Proteins Expressed Early in Alzheimer Disease Mouse Models, Are Essential for the C1q-mediated Protection against Amyloid-β Neurotoxicity
Journal of Biological Chemistry·2012· 159 citedOpen Access
C1q induces a program of gene expression in amyloid beta injured neurons necessary for neuron survival
Immunobiology·2012
Modulation of beta-amyloid aggregation by engineering the sequence connecting beta-strand forming domains
2012
Modulation of beta-amyloid aggregation by engineering the sequence connecting beta-strand forming domains
Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics·2012· 7 cited
Macromolecular crowding in biomatrix
2012· 2 cited
Creation of Aggregation‐Defective α‐Synuclein Variants by Engineering the Sequence Connecting β‐Strand‐Forming Domains
ChemBioChem·2011· 10 cited
Crocetin inhibits beta-amyloid fibrillization and stabilizes beta-amyloid oligomers
Biochemical and Biophysical Research Communications·2011· 44 cited
A Strategy for Designing a Peptide Probe for Detection of β‐Amyloid Oligomers
ChemBioChem·2010· 43 cited
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Frequent Co-authors

Andrea J. Tenner(11), María Isabel Fonseca(8), Shu‐Hui Chu(6), Melody Fang(6), Jin Ryoun Kim(6), Tracy Cole(4), Lindsay A. Hohsfield(4), M.D. Martínez del Valle Torres(4), Kim N. Green(4), Rick A. Wetsel(4), A Mortazavi(4), Yang Hu(4), Baihao Su(3), Lila Modarresi(2), Marie Benoit(2), Minhan L. Dinh(2), Francisca Benavente(2), Osvaldo Vasquez(2), Shan Jiang(2), Agueda Rostagno(2)