Nancy D. Dalton

University of California, San Diego — United States

Medicine · Health Sciences

55h-index13.5kcitations157works1.02yr avg
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Research Topics

Cardiomyopathy and Myosin Studies(53), Cardiovascular Effects of Exercise(32), Cardiac electrophysiology and arrhythmias(20), Muscle Physiology and Disorders(15), Viral Infections and Immunology Research(14)

Publications156 total

Combined Loss of Obsc and Obsl1 in Murine Hearts Results in Diastolic Dysfunction, Altered Metabolism, and Deregulated Mitophagy
Circulation Heart Failure·2025· 3 citedOpen Access
Improvement of hypertrophic cardiomyopathy in knock-in murine model with neonatal gene therapy
Molecular Genetics and Metabolism·2024
Exploring Pompe disease: Insights into the natural history of novel knock-in murine model
Molecular Genetics and Metabolism·2024
CRISPR-mediated generation and characterization of a Gaa homozygous c.1935C>A (p.D645E) Pompe disease knock-in mouse model recapitulating human infantile onset-Pompe disease
Scientific Reports·2022· 10 citedOpen Access
Comparative molecular analysis of two HFpEF/diastolic dysfunction models – in search of a unifying disease mechanism
Journal of Molecular and Cellular Cardiology·2022
Combined loss of obscurin and obscurin-like 1 in murine hearts results in diastolic dysfunction, altered metabolism and deregulated mitophagy
2022· 1 citedOpen Access
CRISPR-Mediated Generation and Characterization of a Gaa Homozygous c.1935C>A (p.D645E) Pompe Disease Knock-in Mouse Model Recapitulates Human Infantile Onset-Pompe Disease
Research Square (Research Square)·2022Open Access
CRISPR-Mediated Generation and Characterization of a <i>Gaa</i> Homozygous c.1935C&gt;A (p.D645E) Pompe Disease Knock-in Mouse Model Recapitulates Human Infantile Onset-Pompe Disease
2022Open Access
CRISPR-mediated generation and characterization of the homozygous c.1935c &gt; a (p.d645e) Pompe disease knock-in mouse model
Molecular Genetics and Metabolism·2022
Abstract 105: Novel Functions For Synaptosomal-associated Protein 29 (snap29) In Cardiac Arrhythmias
Circulation Research·2021
Abstract MP218: Cop9 Signalosome Subunit 6 Restricts Desmosomal Proteome Degradation To Prevent Desmosomal Targeted Cardiac Disease
Circulation Research·2021
Constitutive protein kinase G activation exacerbates stress‐induced cardiomyopathy
British Journal of Pharmacology·2021· 11 citedOpen Access
SNAP29 Restricts Cardiac Arrhythmias by Insulating A Subset of Desmosomal Proteins and Connexin43 from Autophagic Degradation
The FASEB Journal·2021· 1 cited
Desmosomal COP9 regulates proteome degradation in arrhythmogenic right ventricular dysplasia/cardiomyopathy
The FASEB Journal·2021
Desmosomal COP9 regulates proteome degradation in arrhythmogenic right ventricular dysplasia/cardiomyopathy
Journal of Clinical Investigation·2021· 24 citedOpen Access
Delta-Sarcoglycan Gene Therapy Halts Progression of Cardiac Dysfunction, Improves Respiratory Failure, and Prolongs Life in Myopathic Hamsters
UNC Libraries·2020Open Access
CRISPR-Cas9 generated Pompe knock-in murine model exhibits early-onset hypertrophic cardiomyopathy and skeletal muscle weakness
Scientific Reports·2020· 22 citedOpen Access
Cardiomyocyte Expression of ZO-1 Is Essential for Normal Atrioventricular Conduction but Does Not Alter Ventricular Function
Circulation Research·2020· 18 citedOpen Access
Mechanistic Insights Into RNA Splicing As A Trigger For Arrhythmogenic Right Ventricular Cardiomyopathy
The FASEB Journal·2020
Cardiomyocyte SNAP29 Insulates Desmosomes from Targeted Protein Degradation by Selective Autophagy to Restrict Arrhythmias
The FASEB Journal·2020
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Frequent Co-authors

Kirk L. Peterson(30), Yusu Gu(26), Ju Chen(16), John Ross(8), Sylvia Μ. Evans(7), Joan Heller Brown(7), Kenneth R. Chien(6), Kunfu Ouyang(6), Gianluigi Condorelli(5), Donald M. Bers(5), Kirk U. Knowlton(5), Daniele Catalucci(4), Nuno Guimarães‐Camboa(4), Tong Zhang(4), Hongqiang Cheng(4), John Ross(4), David M. Roth(4), Stephan Lange(4), Toshitaka Yajima(4), Andrew D. McCulloch(4)