Max L. Pöhlmann

Harvard University — United States

Neuroscience · Life Sciences

14h-index1.1kcitations25works19.02yr avg
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Research Topics

Stress Responses and Cortisol(11), Pancreatic function and diabetes(6), Autophagy in Disease and Therapy(5), Adipose Tissue and Metabolism(5), Tryptophan and brain disorders(4)

Publications25 total

Inhibiting Hippo pathway kinases releases WWC1 to promote AMPAR-dependent synaptic plasticity and long-term memory in mice
Science Signaling·2024· 14 citedOpen Access
SKA2 regulated hyperactive secretory autophagy drives neuroinflammation-induced neurodegeneration
Nature Communications·2024· 24 citedOpen Access
SKA2 regulated hyperactive secretory autophagy drives neuroinflammation-induced neurodegeneration
2023Open Access
Mediobasal hypothalamic FKBP51 acts as a molecular switch linking autophagy to whole-body metabolism
Science Advances·2022· 24 citedOpen Access
Mediobasal hypothalamic FKBP51 acts as a molecular switch linking autophagy to whole-body metabolism - Dataset IC
Zenodo (CERN European Organization for Nuclear Research)·2021Open Access
Mediobasal hypothalamic FKBP51 acts as a molecular switch linking autophagy to whole-body metabolism - Dataset IC
Zenodo (CERN European Organization for Nuclear Research)·2021Open Access
Mediobasal hypothalamic FKBP51 acts as a molecular switch linking autophagy to whole-body metabolism - Dataset Bz
Zenodo (CERN European Organization for Nuclear Research)·2021Open Access
Mediobasal hypothalamic FKBP51 acts as a molecular switch linking autophagy to whole-body metabolism - Dataset Bz
Zenodo (CERN European Organization for Nuclear Research)·2021Open Access
FKBP51 in the Oval Bed Nucleus of the Stria Terminalis Regulates Anxiety-Like Behavior
eNeuro·2021· 21 citedOpen Access
Stress-primed secretory autophagy promotes extracellular BDNF maturation by enhancing MMP9 secretion
Nature Communications·2021· 122 citedOpen Access
Mediobasal hypothalamic FKBP51 acts as a molecular switch linking autophagy to whole-body metabolism
2021· 3 citedOpen Access
The co-chaperone Fkbp5 shapes the acute stress response in the paraventricular nucleus of the hypothalamus of male mice
Molecular Psychiatry·2021· 105 citedOpen Access
Single-cell molecular profiling of all three components of the HPA axis reveals adrenal ABCB1 as a regulator of stress adaptation
Science Advances·2021· 85 citedOpen Access
Hippo-Released WWC1 Primes Ampa Receptor Regulatory Complexes for Hippocampal Learning
SSRN Electronic Journal·2021Open Access
The stress susceptibility factor FKBP51 controls S-ketamine-evoked release of mBDNF in the prefrontal cortex of mice
Neurobiology of Stress·2020· 28 citedOpen Access
Stress-primed secretory autophagy drives extracellular BDNF maturation
2020· 4 citedOpen Access
Unraveling the functional contribution of Fkbp5 to stress vulnerability
Electronic Theses of LMU Munich (Ludwig-Maximilians-Universität München)·2020Open Access
The co-chaperone Fkbp5 shapes the acute stress response in the paraventricular nucleus of the hypothalamus
2019· 4 citedOpen Access
Pharmacological Modulation of the Psychiatric Risk Factor FKBP51 Alters Efficiency of Common Antidepressant Drugs
Frontiers in Behavioral Neuroscience·2018· 38 citedOpen Access
Chronic CRH depletion from GABAergic, long-range projection neurons in the extended amygdala reduces dopamine release and increases anxiety
Nature Neuroscience·2018· 142 citedOpen Access
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Frequent Co-authors

Mathias V. Schmidt(24), Kathrin Hafner(16), Nils C. Gassen(16), Alexander S. Häusl(15), Alon Chen(14), Thomas Bajaj(12), Jakob Hartmann(10), Frederik Dethloff(9), Lea M. Brix(9), Jan M. Deussing(8), Georgia Balsevich(8), Kerry J. Ressler(6), Elmira Anderzhanova(6), Felix Hausch(6), Silvia Martinelli(6), Clara Engelhardt(6), Meri De Angelis(6), Joachim Nagler(6), Karl‐Werner Schramm(6), Patrick Giavalisco(6)