Paul W. Brandt‐Rauf

Drexel University — United States

Medicine · Health Sciences

39h-index5.5kcitations271works0.72yr avg
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Research Topics

Cancer-related Molecular Pathways(60), Carcinogens and Genotoxicity Assessment(38), Cancer Research and Treatments(26), Occupational and environmental lung diseases(20), Cell death mechanisms and regulation(16)

Publications271 total

JOEM Editorial
Journal of Occupational and Environmental Medicine·2025
HDM-2-Targeting Peptide PNC-27 Kills Cervical Cancer Cells but not Normal Cervical Cells.
PubMed·2025
JOEM Updates and Thank You to the 2024 Reviewers
Journal of Occupational and Environmental Medicine·2025
Occupational Health and Neuroergonomics
Journal of Occupational and Environmental Medicine·2024· 4 cited
JOEM Updates and Thank You to the 2023 Reviewers
Journal of Occupational and Environmental Medicine·2024
Priorities for development of research methods in occupational cancer.
UNC Libraries·2024Open Access
Supplementary Table 1 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure 1 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure 2 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Methods from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure 3 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure Legends from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure 2 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure 4 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure Legends from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure 6 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Table 1 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Data from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure 6 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
Supplementary Figure 5 from Redirecting Apoptosis to Aponecrosis Induces Selective Cytotoxicity to Pancreatic Cancer Cells through Increased ROS, Decline in ATP Levels, and VDAC
2023Open Access
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Frequent Co-authors

Matthew R. Pincus(13), Robert L. Fine(8), Walter P. Carney(7), Kari Hemminki(6), Joseph H. Graziano(6), Ramon Rosal(6), Steven J. Smith(6), Habibul Ahsan(5), Heikki Koskinen(5), Immaculata De Vivo(4), Yu Chen(4), Victor Adler(4), Fred K. Friedman(4), Vesna Slavkovich(4), Josef Michl(4), Harri Vainio(4), Marie‐Jeanne Marion(4), Regina M. Santella(3), Faruque Parvez(3), Ze’ev A. Ronai(3)