Hannah Scheiblich

Max Planck Institute for Biology of Ageing — Germany

Neuroscience · Life Sciences

18h-index3.2kcitations31works102.82yr avg
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Research Topics

Neuroinflammation and Neurodegeneration Mechanisms(17), Alzheimer's disease research and treatments(13), Inflammasome and immune disorders(4), Tryptophan and brain disorders(4), Neurobiology and Insect Physiology Research(3)

Publications31 total

Mobile Powerhouses: Mitochondria transfer via Tunneling Nanotubes in brain health and neurodegenerative diseases
2025Open Access
Metabolic Crossroads: AMDHD2 Couples the Hexosamine Biosynthetic Pathway to Acetyl-CoA Homeostasis in Pluripotent Stem Cells
2025Open Access
Neuroinflammation in Alzheimer disease
Nature reviews. Immunology·2024· 289 citedOpen Access
Microglia rescue neurons from aggregate-induced neuronal dysfunction and death through tunneling nanotubes
Neuron·2024· 122 citedOpen Access
Characterizing microglial senescence: Tau as a key player
Journal of Neurochemistry·2023· 29 citedOpen Access
Microglial NLRP3 Inflammasome Activation upon TLR2 and TLR5 Ligation by Distinct α-Synuclein Assemblies
The Journal of Immunology·2021· 114 citedOpen Access
Microglia jointly degrade fibrillar alpha-synuclein cargo by distribution through tunneling nanotubes
Cell·2021· 320 citedOpen Access
Differential interaction with <scp>TREM2</scp> modulates microglial uptake of modified Aβ species
Glia·2021· 18 citedOpen Access
Nitrergic modulation of neuronal excitability in the mouse hippocampus is mediated via regulation of Kv2 and voltage‐gated sodium channels
Hippocampus·2021· 6 citedOpen Access
Inhibition of neuroinflammatory nitric oxide signaling suppresses glycation and prevents neuronal dysfunction in mouse prion disease
Proceedings of the National Academy of Sciences·2021· 55 citedOpen Access
Innate immune activation of the NLRP3 inflammasome pathway drives tau pathology
Alzheimer s & Dementia·2020
Inhibition of neuroinflammatory nitric oxide signalling supresses protein glycation and recovers neuronal dysfunction in prion disease
Research Square (Research Square)·2020Open Access
Neuroimmune Connections in Aging and Neurodegenerative Diseases
Trends in Immunology·2020· 162 cited
β-Amyloid Clustering around ASC Fibrils Boosts Its Toxicity in Microglia
Cell Reports·2020· 183 citedOpen Access
Microglial NLRP3 Inflammasome Activation Upon TLR2 and TLR5 Ligation by Distinct α-Synuclein Assemblies
SSRN Electronic Journal·2020· 3 citedOpen Access
NLRP3 inflammasome activation drives tau pathology
Nature·2019· 1.4k citedOpen Access
Amyloid β Clustering Around ASC Fibrils Boost Its Toxicity in Microglia
SSRN Electronic Journal·2019· 1 citedOpen Access
Alterations in neuronal metabolism contribute to the pathogenesis of prion disease
Cell Death and Differentiation·2018· 34 citedOpen Access
Transcriptome analysis of alcohol-treated microglia reveals downregulation of beta amyloid phagocytosis
Journal of Neuroinflammation·2018· 53 citedOpen Access
Additional file 1: of Transcriptome analysis of alcohol-treated microglia reveals downregulation of beta amyloid phagocytosis
Figshare·2018Open Access
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Frequent Co-authors

Michael T. Heneka(14), Eicke Latz(8), Gerd Bicker(8), Stephanie Schwartz(7), Angelika Griep(6), Joern R. Steinert(5), Christina Ising(4), Susanne V. Schmidt(4), Ronald Melki(4), Michael Stern(4), Carmen Venegas(3), Róisín M. McManus(3), Francesco Santarelli(3), Frederic Brosseron(3), Sabine Opitz(3), Douglas T. Golenbock(3), Luc Bousset(3), Lena Wischhof(3), Douglas L. Feinstein(3), Julie-Myrtille Bourgognon(3)