Hans Lassmann

Medical University of Vienna — Austria

Medicine · Health Sciences

165h-index96.2kcitations898works14.02yr avg
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Research Topics

Multiple Sclerosis Research Studies(342), Neuroinflammation and Neurodegeneration Mechanisms(190), Peripheral Neuropathies and Disorders(133), Neurogenesis and neuroplasticity mechanisms(88), Immunotherapy and Immune Responses(78)

Publications895 total

The dynamics of brain T cell populations during the course of rasmussen encephalitis: from expansion to exhaustion
Journal of Neuroinflammation·2025· 4 citedOpen Access
Myelin antigen-specific effector CD8 <sup>+</sup> T cells induce chronic CNS autoimmunity in a CD4 <sup>+</sup> T cell-dependent manner
2025Open Access
Subcortical plaques and inflammation reflect cortical and meningeal pathologies in progressive multiple sclerosis
Brain Pathology·2024· 5 citedOpen Access
Microglia regulate cortical remyelination via TNFR1-dependent phenotypic polarization
Cell Reports·2024· 9 citedOpen Access
The influence of MOGAD on diagnosis of multiple sclerosis using MRI
Nature Reviews Neurology·2024· 18 citedOpen Access
Immune-mediated disorders
2024· 2 cited
Demyelinating diseases
2024· 2 cited
Different Complement Activation Patterns Following C5 Cleavage in MOGAD and AQP4-IgG+NMOSD
Neurology Neuroimmunology & Neuroinflammation·2024· 17 citedOpen Access
The ageing central nervous system in multiple sclerosis: the imaging perspective
Brain·2024· 37 citedOpen Access
Issue Information
Annals of Neurology·2024· 1 citedOpen Access
The shoulders we keep standing on: remembering Otto Marburg, a big brain in neurology and multiple sclerosis, at 150
Acta Neuropathologica·2024· 2 citedOpen Access
Determinants and Biomarkers of Progression Independent of Relapses in Multiple Sclerosis
Annals of Neurology·2024· 69 citedOpen Access
New insights into neuropathology and pathogenesis of autoimmune glial fibrillary acidic protein meningoencephalomyelitis
Acta Neuropathologica·2024· 36 citedOpen Access
Ineffective control of Epstein–Barr virus infection is seen in MS: What is next?
Clinical and Translational Medicine·2024· 4 citedOpen Access
IKKβ deletion from CNS macrophages increases neuronal excitability and accelerates the onset of EAE, while from peripheral macrophages reduces disease severity
Journal of Neuroinflammation·2024· 4 citedOpen Access
The relation between <scp>BTK</scp> expression and iron accumulation of myeloid cells in multiple sclerosis
Brain Pathology·2024· 14 citedOpen Access
Microfibrillar-associated protein 4 as a potential marker of acute relapse in inflammatory demyelinating diseases of the central nervous system: Pathological and clinical aspects
Multiple Sclerosis Journal·2023· 3 citedOpen Access
Paramagnetic rim lesions lead to pronounced diffuse periplaque white matter damage in multiple sclerosis
Multiple Sclerosis Journal·2023· 19 citedOpen Access
Microfibrillar-associated Protein 4 as Potential Marker of Acute Relapse in Inflammatory Demyelinating Diseases of the Central Nervous System: Pathological and Clinical Aspects
Research Square (Research Square)·2023Open Access
Lesional Antibody Synthesis and Complement Deposition Associate With De Novo Antineuronal Antibody Synthesis After Spinal Cord Injury
Neurology Neuroimmunology & Neuroinflammation·2023· 11 citedOpen Access
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Frequent Co-authors

Wolfgang Brück(16), Claudia F. Lucchinetti(14), Joseph E. Parisi(8), Christopher Linington(5), Monika Bradl(5), Moses Rodriguez(4), Christine Stadelmann(4), Manfred Schmidbauer(4), Josa M. Frischer(4), Hartmut Wekerle(4), Reinhard Hohlfeld(4), Jan Bauer(4), Bernd W. Scheithauer(3), Helmut Rauschka(3), Stephen D. Weigand(3), Martin Kerschensteiner(3), Don Mahad(3), K. A. Jellinger(3), Lukas Haider(3), Simon Hametner(3)