Frederick Klauschen

LMU Klinikum — Germany

Biochemistry, Genetics and Molecular Biology · Life Sciences

62h-index25.2kcitations453works2.62yr avg
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Research Topics

Cancer Genomics and Diagnostics(134), Cancer Immunotherapy and Biomarkers(70), Lung Cancer Treatments and Mutations(58), Radiomics and Machine Learning in Medical Imaging(42), AI in cancer detection(41)

Publications451 total

Association of recurrence patterns and outcome with HR and HER2 status in patients with resected brain metastases from breast cancer.
PubMed·2026
TP53 mutational status correlates with PD-L1 and L1CAM expression in uterine serous carcinoma
International Journal of Gynecological Cancer·2026
Integrating UHPLC-MS and MALDI-MSI for Spatial Nucleoside Profiling in FFPE Breast Cancer: A Multimodal Molecular Pathology Framework
2026Open Access
Atlas 2 -- Foundation models for clinical deployment
ArXiv.org·2026Open Access
Atlas 2 -- Foundation models for clinical deployment
arXiv (Cornell University)·2026Open Access
[18F]F-FAPI-74 PET/CT for gonadal and extragonadal germ cell Tumors: a pilot study
EJNMMI Research·2025Open Access
Multiregional Immune Profiling Reveals Prognostic Patterns in Bladder Cancer
European Urology Oncology·2025Open Access
Digital Spatial Pathway Mapping Reveals Prognostic Tumor States in Head and Neck Cancer
bioRxiv (Cold Spring Harbor Laboratory)·2025Open Access
Supplementary video file from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
2025Open Access
Supplementary table S4 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
2025Open Access
Supplementary Data from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
2025Open Access
Supplementary table S2 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
2025Open Access
Supplementary table S1 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
2025Open Access
Supplementary Data from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
2025Open Access
Supplementary table S3 from SMAD4 Loss Induces c-MYC–Mediated NLE1 Upregulation to Support Protein Biosynthesis, Colorectal Cancer Growth, and Metastasis
2025Open Access
Individual Patient Data Meta-Analysis of Consensus Molecular Subtypes as Biomarkers of First-Line Treatment in <i>RAS</i> Wild-Type Metastatic Colorectal Cancer
Journal of Clinical Oncology·2025· 1 cited
AI-powered spatial cell phenomics enhances risk stratification in non-small cell lung cancer
Nature Communications·2025· 3 citedOpen Access
MeDi: Metadata-Guided Diffusion Models for Mitigating Biases in Tumor Classification
Lecture notes in computer science·2025Open Access
Correction: Precision Oncology in Rare Endocrine and Neuroendocrine Neoplasms: Experiences and Challenges of the CCCMunichLMU Molecular Tumor Board
Targeted Oncology·2025Open Access
Large Language Models Improve Cancer Survival Prediction Using Real-World Clinical Notes
2025Open Access
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Frequent Co-authors

Carsten Denkert(18), Stephan Wienert(15), Albrecht Stenzinger(13), Sibylle Loibl(11), Jan Budczies(11), Wilko Weichert(11), Manfred Dietel(8), Ronald N. Germain(8), Michael Bockmayr(8), Sherene Loi(6), Peter Schirmacher(6), Giancarlo Pruneri(5), David L. Rimm(5), Christos Sotiriou(5), Giuseppe Viale(5), Sunil Badve(5), Gϋnter von Minckwitz(5), Silvia Darb‐Esfahani(5), Arne Warth(5), Marc Bajénoff(5)