Lev Becker

University of Chicago — United States

Immunology and Microbiology · Life Sciences

30h-index8.6kcitations90works3.92yr avg
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Research Topics

Immune cells in cancer(23), Adipokines, Inflammation, and Metabolic Diseases(16), Adipose Tissue and Metabolism(10), Cancer Mechanisms and Therapy(9), Lipoproteins and Cardiovascular Health(7)

Publications90 total

Developing a therapeutic elastase that stimulates anti-tumor immunity by selectively killing cancer cells
Cell Reports Medicine·2025Open Access
Abstract 861: N17350 outperforms cytotoxic agents in killing tumors of treatment-naïve and chemotherapy-treated ovarian carcinoma patients
Cancer Research·2025
Dynamic investigation of hypoxia-induced L-lactylation
Proceedings of the National Academy of Sciences·2025· 25 citedOpen Access
1297 NEU-002 leverages a cancer specific innate immune pathway to induce immunogenic cell death and stimulate anti-tumor immunity
Regular and Young Investigator Award Abstracts·2024Open Access
Context-dependent roles for autophagy in myeloid cells in tumor progression
2024· 1 citedOpen Access
Abstract 6578: N17465, a systemically deliverable elastase, attenuates tumorigenesis and stimulates anti-tumor immunity
Cancer Research·2024· 2 cited
Abstract 2686: Glycolysis-stimulated lactate production by tumor-associated macrophages promotes their M2-like phenotype
Cancer Research·2024· 2 cited
Abstract 5895: N17350 kills cancer cells, spares immune cells, and regresses CDX tumors from chemotherapy naive and experienced patients
Cancer Research·2024· 1 cited
1344 N17350 combines selective cancer killing with adaptive immune activation to eradicate tumors
Regular and Young Investigator Award Abstracts·2023Open Access
POS0017 ACTIVATED B CELLS ARE ENRICHED FOR RANKL AND PROINFLAMMATORY CYTOKINE PRODUCTION PRIOR TO ONSET OF CLINICAL RHEUMATOID ARTHRITIS
2023Open Access
Abstract 6390: N17350 is an emerging therapeutic modality that selectively kills cancer cells and stimulates anti-tumor immunity
Cancer Research·2023· 2 cited
Supplementary Tables 1 and 2, Figures S1-16 from Metastasis Suppressors Regulate the Tumor Microenvironment by Blocking Recruitment of Prometastatic Tumor-Associated Macrophages
2023Open Access
Data from Metastasis Suppressors Regulate the Tumor Microenvironment by Blocking Recruitment of Prometastatic Tumor-Associated Macrophages
2023Open Access
Supplementary Methods from Metastasis Suppressors Regulate the Tumor Microenvironment by Blocking Recruitment of Prometastatic Tumor-Associated Macrophages
2023Open Access
Supplementary Methods from Metastasis Suppressors Regulate the Tumor Microenvironment by Blocking Recruitment of Prometastatic Tumor-Associated Macrophages
2023Open Access
Supplementary Tables 1 and 2, Figures S1-16 from Metastasis Suppressors Regulate the Tumor Microenvironment by Blocking Recruitment of Prometastatic Tumor-Associated Macrophages
2023Open Access
Data from Metastasis Suppressors Regulate the Tumor Microenvironment by Blocking Recruitment of Prometastatic Tumor-Associated Macrophages
2023Open Access
Abstract F2-2: Neutrophil elastase and anti-tumor effects
Cancer Research·2023
An Iron Refractory Phenotype in Obese Adipose Tissue Macrophages Leads to Adipocyte Iron Overload
International Journal of Molecular Sciences·2022· 18 citedOpen Access
Modifying Macrophage Phenotypes to Dampen Airway Inflammation in Chronic Lung Diseases
2022
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Frequent Co-authors

Kelly Q. Schoenfelt(16), Chang Cui(13), Marlys L. Koschinsky(11), Tomáš Vaisar(8), Jay W. Heinecke(8), Karin Bornfeldt(6), Kasturi Chakraborty(5), Guolin Zhou(5), Catherine A. Reardon(5), Daniel M. Czyż(4), Howard A. Shuman(4), Katherine B. Hisert(4), Alexandria Hoffman(4), Ariane Blank(4), Michelle M. Averill(4), Michael E. Nesheim(4), Yamuna Krishnan(4), Sina A. Gharib(4), Guolin Zhou(3), Brittney R. Coats(3)