Parmjit Jat

MRC Prion Unit — United Kingdom

Medicine · Health Sciences

50h-index9.9kcitations174works3.02yr avg
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Research Topics

Telomeres, Telomerase, and Senescence(23), Virus-based gene therapy research(19), Cancer-related Molecular Pathways(14), Polyomavirus and related diseases(14), Pharmaceutical studies and practices(14)

Publications174 total

A dividing cell model for stable propagation and curing of <i>bona fide</i> human sporadic Creutzfeldt-Jakob Disease prions
bioRxiv (Cold Spring Harbor Laboratory)·2025Open Access
Endothelial Cells Differentiated from Human Induced Pluripotent Stem Cells Form Aligned Network Structures in Engineered Neural Tissue
Journal of Functional Biomaterials·2025Open Access
Engineered Neural Tissue (EngNT) Containing Human iPSC-Derived Schwann Cell Precursors Promotes Axon Growth in a Rat Model of Peripheral Nerve Injury
Bioengineering·2025· 1 citedOpen Access
Corrigendum to “Prion Propagation is Dependent on Key Amino Acids in Charge Cluster 2 within the Prion Protein” [J. Mol. Biol. 435(4) (2023) 167925]
Journal of Molecular Biology·2025Open Access
B7-H3 Expression in Breast Cancer and Brain Metastasis
International Journal of Molecular Sciences·2024· 14 citedOpen Access
Tumor heterogeneity in VHL drives metastasis in clear cell renal cell carcinoma
Signal Transduction and Targeted Therapy·2023· 66 citedOpen Access
Supplementary Data from Decreased Poly(ADP-Ribosyl)ation of CTCF, a Transcription Factor, Is Associated with Breast Cancer Phenotype and Cell Proliferation
2023Open Access
Data from Decreased Poly(ADP-Ribosyl)ation of CTCF, a Transcription Factor, Is Associated with Breast Cancer Phenotype and Cell Proliferation
2023Open Access
Data from Decreased Poly(ADP-Ribosyl)ation of CTCF, a Transcription Factor, Is Associated with Breast Cancer Phenotype and Cell Proliferation
2023Open Access
Supplementary Data from Decreased Poly(ADP-Ribosyl)ation of CTCF, a Transcription Factor, Is Associated with Breast Cancer Phenotype and Cell Proliferation
2023Open Access
Supplementary Table 4 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Supplementary Figure Legends 1-2 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Supplementary Table 3 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Supplementary Table 2 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Supplementary Table 5 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Supplementary Figure 2 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Supplementary Table 3 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Data from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Supplementary Figure 1 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
Supplementary Figure 1 from Ability to Acquire Drug Resistance Arises Early during the Tumorigenesis Process
2023Open Access
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Frequent Co-authors

Mark Noble(8), Phillip A. Sharp(6), Paris Ataliotis(4), Michael J. O’Hare(4), Alan Nevill(4), Holger Hummerich(4), Alan Mackay(3), Sunil R. Lakhani(3), Maria Leao(3), John Collinge(3), Thomas M. Roberts(3), Ole Gjoerup(3), Sarah J. Tabrizi(2), Constance L. Cepko(2), Richard C. Mulligan(2), Peter T. Simpson(2), Anita Grigoriadis(2), Andrew J.G. Simpson(2), Parineeta Arora(2), Yuan Cheng(2)