Simon S. McDade

Queen's University Belfast — United Kingdom

Biochemistry, Genetics and Molecular Biology · Life Sciences

31h-index2.9kcitations350works0.52yr avg
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Research Topics

Cancer, Lipids, and Metabolism(53), Prostate Cancer Treatment and Research(45), Cancer Genomics and Diagnostics(44), Cancer Research and Treatments(43), Pancreatic and Hepatic Oncology Research(39)

Publications348 total

Figure 6 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Figure 4 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Figure 5 from Functional Genomic Identification of Predictors of Sensitivity and Mechanisms of Resistance to Multivalent Second-Generation TRAIL-R2 Agonists
2025Open Access
Supplementary Data 1 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Figure 4 from Functional Genomic Identification of Predictors of Sensitivity and Mechanisms of Resistance to Multivalent Second-Generation TRAIL-R2 Agonists
2025Open Access
Supplementary Figures S1-S6 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Figure 2 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Supplementary Materials and Methods SM1 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Figure 5 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Supplementary Data 2 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Figure 1 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Figure 3 from Bcl-xL Is a Key Mediator of Apoptosis Following KRAS<sup>G12C</sup> Inhibition in <i>KRAS<sup>G12C</sup></i>-mutant Colorectal Cancer
2025Open Access
Figure 3 from Functional Genomic Identification of Predictors of Sensitivity and Mechanisms of Resistance to Multivalent Second-Generation TRAIL-R2 Agonists
2025Open Access
Figure 2 from Functional Genomic Identification of Predictors of Sensitivity and Mechanisms of Resistance to Multivalent Second-Generation TRAIL-R2 Agonists
2025Open Access
Figure 1 from Functional Genomic Identification of Predictors of Sensitivity and Mechanisms of Resistance to Multivalent Second-Generation TRAIL-R2 Agonists
2025Open Access
Spatial-Immune Multi-omics Refines Prognostication in Early-Stage Estrogen Receptor-Positive Breast Cancer
2025Open Access
High-Resolution Spatial Proteomics Characterises Colorectal Cancer Consensus Molecular Subtypes
2025· 3 citedOpen Access
Abstract 161: High-resolution spatial proteomics characterizes colorectal cancer consensus molecular subtypes
Cancer Research·2025
Understanding Ovarian Cancer: Lessons Learned From Cells, Apps And Patients
International Journal of Gynecological Cancer·2025
Table S5 from Ruthenium Drug BOLD-100 Regulates <i>BRAF</i>MT Colorectal Cancer Cell Apoptosis through AhR/ROS/ATR Signaling Axis Modulation
2024Open Access
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Frequent Co-authors

Dennis J. McCance(14), Daniel B. Longley(13), Mark Lawler(8), Nyree Crawford(7), Philip D. Dunne(7), Adam Pickard(6), Tamas Sessler(6), Darragh G. McArt(5), David Waugh(5), Patrick G. Johnston(5), Manuel Salto‐Tellez(5), Jacqueline A. James(5), Christopher J. Lord(5), D. Patel(5), Christopher McCann(5), Maurice B. Loughrey(4), Wendy L. Allen(4), D. Paul Harkin(4), Stephanie G. Craig(4), Michael Moran(4)