Andrew P. Feinberg

Johns Hopkins University — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

120h-index97.8kcitations475works0.72yr avg
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Research Topics

Epigenetics and DNA Methylation(293), Genetic Syndromes and Imprinting(123), Pancreatic and Hepatic Oncology Research(69), Cancer Genomics and Diagnostics(59), Prenatal Screening and Diagnostics(52)

Publications465 total

Supplementary Data from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 7 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 4 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 2 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 3 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 1 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 5 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 6 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
DNMT3A R882H Is Not Required for Disease Maintenance in Primary Human AML but Is Associated with Increased Leukemia Stem Cell Frequency
Cancer Discovery·2025Open Access
Radiation effects research foundation—a view to the future
Carcinogenesis·2025· 1 citedOpen Access
Moderate to Severe Impairment of D <scp>l</scp>  CO and Iso↓D <scp>l</scp>  CO Are Associated with All-Cause Mortality in People with Human Immunodeficiency Virus
Annals of the American Thoracic Society·2025Open Access
Ultra-processed foods, plant and animal sources, and all-cause, cardiovascular, and cancer mortality in older adults in the United States: results from the Cardiovascular Health Study
American Journal of Clinical Nutrition·2025· 3 citedOpen Access
Correction: Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
Cancer Research·2025Open Access
SUBOPTIMAL HYPERTENSION CONTROL IN YOUNG PATIENTS COMPARED TO OLDER PATIENTS IN A LARGE HEALTH SYSTEM
Journal of the American College of Cardiology·2025Open Access
DNA methylation memory of pancreatic acinar-ductal metaplasia transition state altering Kras-downstream PI3K and Rho GTPase signaling in the absence of Kras mutation
Genome Medicine·2025· 2 citedOpen Access
Supplementary Table 6 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Data from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 2 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 7 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
Supplementary Table 5 from Comprehensive DNA Methylation Analysis Indicates That Pancreatic Intraepithelial Neoplasia Lesions Are Acinar-Derived and Epigenetically Primed for Carcinogenesis
2025Open Access
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Frequent Co-authors

Rafael A. Irizarry(11), Bert Vogelstein(8), Martin J. Aryee(7), Akiko Doi(6), Bo Wen(6), Hengmi Cui(6), M. Daniele Fallin(6), Christine Ladd‐Acosta(5), Peter Murakami(5), George Q. Daley(5), Hao Wu(5), Patrick Onyango(4), Sheri A. Brandenburg(4), Andrew E. Jaffe(3), Hong Ji(3), Riccardo E. Marioni(3), Sonia Shah(3), Allan F. McRae(3), Brian H. Chen(3), Allan C. Just(3)