Thomas J. Sproule

Jackson Laboratory — United States

Immunology and Microbiology · Life Sciences

22h-index3.1kcitations66works0.82yr avg
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Research Topics

T-cell and B-cell Immunology(26), Immune Cell Function and Interaction(20), Monoclonal and Polyclonal Antibodies Research(17), Skin and Cellular Biology Research(10), Immunotherapy and Immune Responses(7)

Publications66 total

Author response: JAX Animal Behavior System (JABS): A genetics informed, end-to-end advanced behavioral phenotyping platform for the laboratory mouse
2025Open Access
JAX Animal Behavior System (JABS): A genetics informed, end-to-end advanced behavioral phenotyping platform for the laboratory mouse
2025Open Access
JAX Animal Behavior System (JABS): A genetics informed, end-to-end advanced behavioral phenotyping platform for the laboratory mouse
2025· 1 citedOpen Access
JAX Animal Behavior System (JABS): A genetics informed, end-to-end advanced behavioral phenotyping platform for the laboratory mouse
2025Open Access
Phenotypic differences in intermediate generalized junctional epidermolysis bullosa with homozygous <i>LAMC2</i> mutation and a potential genetic modifier
Journal of the European Academy of Dermatology and Venereology·2024· 3 cited
Dystonin modifiers of junctional epidermolysis bullosa and models of epidermolysis bullosa simplex without dystonia musculorum
PLoS ONE·2023· 2 citedOpen Access
Functional analysis of Collagen 17a1: A genetic modifier of junctional epidermolysis bullosa in mice
PLoS ONE·2023· 6 citedOpen Access
Seven naturally variant loci serve as genetic modifiers of Lamc2jeb induced non-Herlitz junctional Epidermolysis Bullosa in mice
PLoS ONE·2023· 6 citedOpen Access
Seven naturally variant loci serve as genetic modifiers of <i> Lamc2 <sup>jeb</sup> </i> induced non-Herlitz Junctional Epidermolysis Bullosa in mice
2023Open Access
JAX Animal Behavior System (JABS): A genetics informed, end-to-end advanced behavioral phenotyping platform for the laboratory mouse
2022· 7 citedOpen Access
Diverse dystonin gene mutations cause distinct patterns of <i>Dst</i> isoform deficiency and phenotypic heterogeneity in <i>D</i> <i>ystonia musculorum</i> mice
Disease Models & Mechanisms·2020· 11 citedOpen Access
Structural Variant in Mitochondrial-Associated Gene (MRPL3) Induces Adult-Onset Neurodegeneration with Memory Impairment in the Mouse
Journal of Neuroscience·2020· 12 citedOpen Access
Abrogated AID Function Prolongs Survival and Diminishes Renal Pathology in the BXSB Mouse Model of Systemic Lupus Erythematosus
The Journal of Immunology·2020· 9 citedOpen Access
Enhancing the efficacy of glycolytic blockade in cancer cells<i>via</i>RAD51 inhibition
Cancer Biology & Therapy·2018· 14 citedOpen Access
AI-03 Efficacy and safety of intermittent 2-deoxyglucose therapy in mouse models of lupus
2018· 1 citedOpen Access
Abstract A115: Targeting glycolysis and RAD51-dependent repair in B-lymphoid cancers
Molecular Cancer Therapeutics·2018
医学研究実習レポート 単純性表皮水疱症モデルとしての新規Dystonin/Bpag1遺伝子変異マウスの解析
Niigata medical journal·2017
Precocious Interleukin 21 Expression in Naive Mice Identifies a Natural Helper Cell Population in Autoimmune Disease
Cell Reports·2017· 21 citedOpen Access
AI-19 Metabolic inhibition by 2-deoxyglucose prevents and reverses lupus in mice
2016· 4 citedOpen Access
Natural TFH arise in the thymus and periphery of young naive mice
The Journal of Immunology·2016
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Frequent Co-authors

Derry C. Roopenian(46), Gregory J. Christianson(16), Herbert C. Morse(13), Jason A. Bubier(9), John P. Sundberg(7), Elisabeth Adkins(7), Eun Young Choi(6), Giljun Park(6), John J. Wilson(5), Stefka B. Petkova(4), Daniel J. Shaffer(4), Caroline G. McPhee(4), Shweta Jain(4), Xulong Wang(4), Shreeram Akilesh(3), Nadja Jung(3), Oded Foreman(3), Rosanne Spolski(3), Warren J. Leonard(3), Yoshitaka Yoshimura(3)