Nah-Young Shin

Dana-Farber Cancer Institute — United States

Medicine · Health Sciences

8h-index713citations15works0.02yr avg
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Research Topics

Cell Adhesion Molecules Research(6), Cellular Mechanics and Interactions(4), Acute Myeloid Leukemia Research(3), Hippo pathway signaling and YAP/TAZ(3), Glycosylation and Glycoproteins Research(2)

Publications15 total

Tolerance of Whole Genome Duplication in Megakaryocytes, and Consequences for Their Genome Integrity
Blood·2024
Blood Flow Directs Yap/Taz-Mediated Transcriptional Regulation of Self-Renewal Programs to Control Developmental HSPC Expansion By Mechanical Stimulation of Piezo1
Blood·2023· 2 cited
2006 – MECHANISMS OF FLOW-DRIVEN TRANSCRIPTIONAL CONTROL OF HEMATOPOIETIC STEM AND PROGENITOR CELL DEVELOPMENT BY YAP AND TAZ
Experimental Hematology·2023
3017 – MECHANISMS OF FLOW-DRIVEN TRANSCRIPTIONAL CONTROL OF HEMATOPOIETIC STEM CELL DEVELOPMENT VIA YAP/TAZ REGULATION
Experimental Hematology·2022
2004 – GERMLINE ANKRD26 MUTATIONS IN FAMILIAL THROMBOCYTOPENIA AND LEUKEMIA PREDISPOSITION
Experimental Hematology·2022
3005 – ELIMINATION OF RUNX1 MUTANT PRE-LEUKEMIC CLONES IN THERAPY-RELATED MYELODYSPLASTIC SYNDROME
Experimental Hematology·2020
Role of RUNX Family Transcription Factors in DNA Damage Response.
PubMed·2020· 38 citedOpen Access
Dynamin and endocytosis are required for the fusion of osteoclasts and myoblasts
The Journal of Cell Biology·2014· 83 citedOpen Access
Protein Targets of Reactive Electrophiles in Human Liver Microsomes
Chemical Research in Toxicology·2008· 2 citedOpen Access
The Integrin-coupled Signaling Adaptor p130Cas Suppresses Smad3 Function in Transforming Growth Factor-β Signaling
Molecular Biology of the Cell·2008· 49 citedOpen Access
Protein Targets of Reactive Electrophiles in Human Liver Microsomes
Chemical Research in Toxicology·2007· 96 citedOpen Access
Requirement of Nck adaptors for actin dynamics and cell migration stimulated by platelet-derived growth factor B
Proceedings of the National Academy of Sciences·2006· 76 citedOpen Access
Crk-Associated Substrate Tyrosine Phosphorylation Sites Are Critical for Invasion and Metastasis of Src-Transformed Cells
Molecular Cancer Research·2005· 117 citedOpen Access
CAS promotes invasiveness of Src-transformed cells
Oncogene·2004· 93 cited
Mechanisms of CAS Substrate Domain Tyrosine Phosphorylation by FAK and Src
Molecular and Cellular Biology·2001· 157 citedOpen Access

Frequent Co-authors

Alan Cantor(6), Steven K. Hanks(5), Wade W. Sugden(3), Zachary LeBlanc(3), Maria Gonzalez di Tillio(3), Mohamad Najia(3), Caroline Kubaczka(3), Thorsten M. Schlaeger(3), Edroaldo Lummertz da Rocha(3), Stuart H. Orkin(3), Wolfram Goessling(3), George Q. Daley(3), Trista E. North(3), Jan Brábek(2), Sábata S. Constancio(2), Ambra Pozzi(2), Sheryl L. Stamer(2), D.C. Liebler(2), Ran Jing(2), Elizabeth Molnar(2)