Michael P. Coleman

Babraham Institute — United Kingdom

Neuroscience · Life Sciences

61h-index14.1kcitations177works5.02yr avg
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Research Topics

Nerve injury and regeneration(39), Alzheimer's disease research and treatments(34), Sirtuins and Resveratrol in Medicine(34), Neurogenesis and neuroplasticity mechanisms(28), Mitochondrial Function and Pathology(23)

Publications177 total

Inhibiting Glycogen Synthase Kinase 3 Suppresses TDP-43-Mediated Neurotoxicity in a Caspase-Dependent Manner
Molecular Neurobiology·2026Open Access
Tau oligomers can occur in human brains within days of a single traumatic brain injury
2025Open Access
SARM1 activation induces reversible mitochondrial dysfunction and can be prevented in human neurons by antisense oligonucleotides
Neurobiology of Disease·2025· 4 citedOpen Access
Inhibiting glycogen synthase kinase 3 suppresses TDP-43-mediated neurotoxicity in a caspase-dependent manner.
Apollo (University of Cambridge)·2025Open Access
Inhibiting glycogen synthase kinase 3 suppresses TDP-43-mediated neurotoxicity in a caspase-dependent manner
2025Open Access
Chronically Low NMNAT2 Expression Causes Sub-lethal SARM1 Activation and Altered Response to Nicotinamide Riboside in Axons
Molecular Neurobiology·2024· 5 citedOpen Access
Author Correction: NMNAT2 is a druggable target to drive neuronal NAD production
Nature Communications·2024Open Access
NMNAT2 is a druggable target to drive neuronal NAD production
Nature Communications·2024· 26 citedOpen Access
SARM1 activation induces reversible mitochondrial dysfunction and can be prevented in human neurons by antisense oligonucleotides
2024· 5 citedOpen Access
Programmed axon death: a promising target for treating retinal and optic nerve disorders
Eye·2024· 8 citedOpen Access
Lowering glucose enhances BACE1 activity and Aβ generation in mouse brain slice cultures
2024Open Access
Chronically low NMNAT2 expression causes sub-lethal SARM1 activation and altered response to nicotinamide riboside in axons
2024· 2 citedOpen Access
Adaptation of a Commercial NAD+ Quantification Kit to Assay the Base-Exchange Activity and Substrate Preferences of SARM1
Molecules·2024· 1 citedOpen Access
NMNAT2 supports vesicular glycolysis via NAD homeostasis to fuel fast axonal transport
Molecular Neurodegeneration·2024· 19 citedOpen Access
Adaptation of a commercial NAD quantification kit to assay the base exchange activity of SARM1
2023Open Access
Microglia-mediated demyelination protects against CD8+ T cell-driven axon degeneration in mice carrying PLP defects
Nature Communications·2023· 35 citedOpen Access
Schwann cells are axo-protective after injury irrespective of myelination status in mouse Schwann cell–neuron cocultures
Journal of Cell Science·2023· 12 citedOpen Access
Author Reply to Peer Reviews of A method for mouse myelinating Schwann cell-DRG neuron compartmentalised cocultures: myelination status does not influence the axo-protective effect of Schwann cells after injury.
2023
Genetically perturbed myelin as a risk factor for neuroinflammation-driven axon degeneration
2023· 1 citedOpen Access
NMNAT2 supports vesicular glycolysis via NAD homeostasis to fuel fast axonal transport
2023· 1 citedOpen Access
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Frequent Co-authors

Róbert Adalbert(14), Jonathan Gilley(14), Laura Conforti(13), Richard R. Ribchester(11), Bogdan Beirowski(7), Diana Wagner(6), Giulio Magni(6), Lucie Janečková(6), Elisabetta Babetto(6), Giuseppe Orsomando(5), Andrea Loreto(5), Weiqian Mi(4), Derek Thomson(4), Klaus Addicks(4), V. Hugh Perry(4), M. Di Stefano(4), Till G.A. Mack(3), Thomas H. Gillingwater(3), Andrea Tarlton(3), Simon Andrews(3)