David E. Housman

Massachusetts Institute of Technology — United States

Neuroscience · Life Sciences

120h-index64.3kcitations437works7.02yr avg
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Research Topics

Genetic Neurodegenerative Diseases(68), Mitochondrial Function and Pathology(43), Renal and related cancers(38), Cancer Genomics and Diagnostics(32), Genomics and Chromatin Dynamics(26)

Publications436 total

A rare genetic variant confers resistance to neurodegeneration across multiple neurological disorders by augmenting selective autophagy
Neuron·2025· 4 citedOpen Access
CD4 T cells restricted to DRB1*15:01 recognize two Epstein–Barr virus glycoproteins capable of intracellular antigen presentation
Proceedings of the National Academy of Sciences·2024· 10 citedOpen Access
Huntingtin contains an ubiquitin-binding domain and regulates lysosomal targeting of mitochondrial and RNA-binding proteins
Proceedings of the National Academy of Sciences·2024· 7 citedOpen Access
Supplementary Figures 1-7 from ROS Fusion Tyrosine Kinase Activates a SH2 Domain–Containing Phosphatase-2/Phosphatidylinositol 3-Kinase/Mammalian Target of Rapamycin Signaling Axis to Form Glioblastoma in Mice
2023Open Access
Supplementary Figure 10 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 3 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Data from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 1 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 7 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Methods, Figures 1-2, Tables 1-2 from Epigenetic Regulation of c-ROS Receptor Tyrosine Kinase Expression in Malignant Gliomas
2023Open Access
Supplementary Methods, Figures 1-2, Tables 1-2 from Epigenetic Regulation of c-ROS Receptor Tyrosine Kinase Expression in Malignant Gliomas
2023Open Access
Supplementary Figure Legends 1-10 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 9 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 1 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 6 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 7 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 10 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Data from ROS Fusion Tyrosine Kinase Activates a SH2 Domain–Containing Phosphatase-2/Phosphatidylinositol 3-Kinase/Mammalian Target of Rapamycin Signaling Axis to Form Glioblastoma in Mice
2023Open Access
Supplementary Methods from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
Supplementary Figure 8 from Chronic Activation of Wild-Type Epidermal Growth Factor Receptor and Loss of Cdkn2a Cause Mouse Glioblastoma Formation
2023Open Access
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Frequent Co-authors

Alan Buckler(9), Daniel A. Haber(7), Jerry Pelletier(6), J. David Brook(4), Vincent P. Stanton(4), Tom Glaser(4), Katherine M. Call(4), Julian Borrow(4), Scott W. Lowe(3), Tyler Jacks(3), Mila E. McCurrach(3), Ann M. Graybiel(3), Piet Gros(3), James M. Croop(3), Martin Schalling(3), H. Earl Ruley(2), Robert Sohn(2), S.A. Rundle(2), Hiroaki Kawasaki(2), Gregory M. Springett(2)