Takefumi Ishii

Medicine · Health Sciences

15h-index1.2kcitations30works0.02yr avg
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Research Topics

Myeloproliferative Neoplasms: Diagnosis and Treatment(19), Eosinophilic Disorders and Syndromes(15), Chronic Myeloid Leukemia Treatments(14), Kruppel-like factors research(6), T-cell and B-cell Immunology(5)

Publications30 total

Supplementary Figure 1 from Correction of the Abnormal Trafficking of Primary Myelofibrosis CD34<sup>+</sup> Cells by Treatment with Chromatin-Modifying Agents
2023Open Access
Data from Correction of the Abnormal Trafficking of Primary Myelofibrosis CD34<sup>+</sup> Cells by Treatment with Chromatin-Modifying Agents
2023Open Access
Supplementary Figure 1 from Correction of the Abnormal Trafficking of Primary Myelofibrosis CD34<sup>+</sup> Cells by Treatment with Chromatin-Modifying Agents
2023Open Access
Data from Correction of the Abnormal Trafficking of Primary Myelofibrosis CD34<sup>+</sup> Cells by Treatment with Chromatin-Modifying Agents
2023Open Access
[Successful unrelated cord blood transplantation for extensive meningeal juvenile xanthogranuloma developing after treatment of Langerhans cell histiocytosis in a child].
PubMed·2020· 3 cited
Human CD34+ cells are capable of generating normal and JAK2V617F positive endothelial like cells in vivo
Blood Cells Molecules and Diseases·2009· 33 cited
Correction of the Abnormal Trafficking of Primary Myelofibrosis CD34+ Cells by Treatment with Chromatin-Modifying Agents
Cancer Research·2009· 36 cited
Pivotal role of mast cells in pruritogenesis in patients with myeloproliferative disorders
Blood·2009· 81 citedOpen Access
Mast Cells Are Involved by the Malignant Process and Play An Important Role in the Pruritogenesis in Patients with Myeloproliferative Disorders
Blood·2008· 1 cited
Correction of the Abnormal Trafficking of Primary Myelofibrosis CD34+ Cells by Treatment with Chromatin Modifying Agents
Blood·2008· 4 cited
MNX1–ETV6 fusion gene in an acute megakaryoblastic leukemia and expression of the MNX1 gene in leukemia and normal B cell lines
Cancer Genetics and Cytogenetics·2008· 29 cited
Circulating angiogenic monocyte progenitor cells are reduced in JAK2V617F high allele burden myeloproliferative disorders
Blood Cells Molecules and Diseases·2008· 13 cited
Two Classes of Progenitor Cells in Patients with Myeloproliferative Disorders Are Capable of Generating JAK2V617F+CD31+CD144+ Endothelial Cells.
Blood·2007
Behavior of CD34+ cells isolated from patients with polycythemia vera in NOD/SCID mice
Experimental Hematology·2007· 40 citedOpen Access
Effects of Chromatin-Modifying Agents on CD34+ Cells from Patients with Idiopathic Myelofibrosis
Cancer Research·2007· 63 citedOpen Access
Pivotal contributions of megakaryocytes to the biology of idiopathic myelofibrosis
Blood·2007· 210 citedOpen Access
Haematopoietic cell lineage distribution of <i>MPL</i>W515L/K mutations in patients with idiopathic myelofibrosis
British Journal of Haematology·2007· 14 cited
Erlotinib Effectively Inhibits JAK2V617F Activity and Polycythemia Vera Cell Growth
Journal of Biological Chemistry·2006· 122 citedOpen Access
Engraftment of Subpopulations of Hematopoietic Cells from Patients with Polycythemia Vera in NOD/SCID Mice.
Blood·2006
Identification of the Origins and Characterization of the Consequences of Megakaryocytic Hyperplasia in Idiopathic Myelofibrosis.
Blood·2006
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Frequent Co-authors

Mingjiang Xu(19), Ronald Hoffman(15), Selçuk Sözer(12), Jiapeng Wang(10), Xiaoli Wang(8), E Bruno(6), Kohichiro Tsuji(5), Yasuhiro Ebihara(5), Tatsutoshi Nakahata(5), Masahiro Sako(5), Yan Zhao(4), Shigetaka Asano(4), Taira Maekawa(4), Wen‐Yang Hu(3), Ryuhei Tanaka(3), Wei Zhang(3), Nathaniel Wisch(3), Jun Shi(3), Robert S. Hoffman(3), Stefan O. Ciurea(2)