Satya P. Kalra

University of Florida — United States

Neuroscience · Life Sciences

60h-index13.8kcitations201works0.02yr avg
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Research Topics

Regulation of Appetite and Obesity(102), Hypothalamic control of reproductive hormones(79), Neuropeptides and Animal Physiology(72), Biochemical Analysis and Sensing Techniques(46), Neuroendocrine regulation and behavior(34)

Publications200 total

Hypothalamic Leptin Gene Therapy Reduces Bone Marrow Adiposity in ob/ob Mice Fed Regular and High-Fat Diets
Frontiers in Endocrinology·2016· 43 citedOpen Access
Hypothalamic leptin gene therapy reduces body weight without accelerating age-related bone loss
Journal of Endocrinology·2015· 14 citedOpen Access
Leptin Therapy as a Substitute for Insulin Replacement in Experimental Models of Diabetes: Clinical Implications in Humans
2014
Should leptin replace insulin as a lifetime monotherapy for diabetes type 1 and 2?
Indian Journal of Endocrinology and Metabolism·2013· 4 citedOpen Access
Peripheral leptin regulates bone formation
Journal of Bone and Mineral Research·2012· 259 citedOpen Access
Leptin Gene Therapy for Hyperphagia, Obesity, Metabolic Diseases, and Addiction
Oxford University Press eBooks·2012
Hypothalamic leptin gene therapy prevents weight gain without long-term detrimental effects on bone in growing and skeletally mature female rats
Journal of Bone and Mineral Research·2011· 24 citedOpen Access
Update on Ghrelin
International Journal of Peptides·2010· 12 citedOpen Access
Neuroendocrine Control of Energy Homeostasis: Update on New Insights
Progress in brain research·2010· 33 cited
Central leptin gene therapy ameliorates diabetes type 1 and 2 through two independent hypothalamic relays; a benefit beyond weight and appetite regulation
Peptides·2009· 46 citedOpen Access
Leptin increases osteoblast-specific osteocalcin release through a hypothalamic relay
Peptides·2009· 45 citedOpen Access
Central leptin gene therapy, a substitute for insulin therapy to ameliorate hyperglycemia and hyperphagia, and promote survival in insulin-deficient diabetic mice
Peptides·2009· 48 cited
Increased leptin supply to hypothalamus by gene therapy confers life-long benefits on energy homeostasis, disease cluster of metabolic syndrome- diabetes type 1 and 2, dyslipidemia and cardiovascular ailments- and bone remodeling
2009· 3 cited
Inhibition of p66ShcA redox activity in cardiac muscle cells attenuates hyperglycemia-induced oxidative stress and apoptosis
American Journal of Physiology-Heart and Circulatory Physiology·2008· 35 citedOpen Access
Abstract 1390: Enhanced Leptin Expression in the Hypothalamus Reverses p66ShcA Dependent Oxidative Stress and Myocyte Apoptosis in Akita Mice
Circulation·2008
Leptin-ghrelin-hypothalamic crosstalk in the control of diabetes and cardiovascular diseases
日本内分泌学会雑誌·2008
Disruption in the leptin–NPY link underlies the pandemic of diabetes and metabolic syndrome: New therapeutic approaches
Nutrition·2008· 40 citedOpen Access
S2095a Leptin As a Substitute for Insulin Therapy in Insulin-Deficient Diabetes: Correction of Hyperglycemia and Hyperphgia and Surviving Without Diabetic Complications
Gastroenterology·2008
Erratum
Neuroendocrinology·2008· 1 cited
Errata
Neuroendocrinology·2008Open Access
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Frequent Co-authors

Pushpa S. Kalra(29), Michael G. Dube(8), William R. Crowley(8), Abhiram Sahu(7), Tamás L. Horváth(6), John T. Clark(4), Shuye Pu(3), Bin Xu(3), James W. Simpkins(3), Sabrina Diano(3), Naohiko Ueno(3), Frederick Naftolin(2), Russell T. Turner(2), Stéphane Boghossian(2), Urszula T. Iwaniec(2), Michela Bagnasco(2), Christopher A. Leadem(2), Michael G Dube(2), Harveen Dhillon(2), Yan-Ting Zhou(2)