N Qureshi

Aga Khan University — Pakistan

Immunology and Microbiology · Life Sciences

32h-index4.2kcitations63works0.02yr avg
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Research Topics

Immune Response and Inflammation(19), Lipid Membrane Structure and Behavior(17), Fatty Acid Research and Health(10), Metabolomics and Mass Spectrometry Studies(7), Neonatal Respiratory Health Research(6)

Publications63 total

ARTIFICIAL INTELLIGENCE IN EARLY DETECTION OF MYOCARDIAL INFARCTION USING WEARABLE DEVICES: A SYSTEMATIC REVIEW
Insights-Journal of Health and Rehabilitation·2025Open Access
RAFI: Parallel Dynamic Test-suite Reduction for Software
Soft Computing Research Society eBooks·2021· 8 citedOpen Access
Selected Compounds Modulate Various Inflammatory Biomarkers in Lipopolysaccharide-Induced Macrophages of PPAR-α Knockout Mice
Journal of Clinical and Experimental Research in Cardiology·2017· 1 citedOpen Access
HEMORRHAGIC SHOCK IN THE RAT
Shock·2006
ROLE OF INFLAMMATORY TRANSCRIPTION FACTORS ON APOPTOSIS AND CASPASE 7 EXPRESSION FOLLOWING HEMORRHAGIC SHOCK.
Shock·2003
CELLULAR CAVEOLIN-1 PROTEIN EXPRESSION IN LIPOPOLYSACCHARIDE TREATED MURINE MACROPHAGES.
Shock·2003
BACTERIAL CpG DNA ACTIVATES TRANSCRIPTION FACTOR C/EBP-DELTA IN MOUSE MACROPHAGES.
Shock·2003
BACTERIAL LIPOPOLISACCHARIDE SUPPRESSES EXPRESSION OF DNA METHYLTRANSFERASE 1 IN MOUSE MACROPHAGES.
Shock·2003
A NOVEL LPS-ASBA PHOTOACTIVE CROSSLINKING AGENT BINDS TO C2 and N3 SUBUNITS OF MACROPHAGE PROTEASOME. LPS DIFFERENTIALLY ACTIVATES THE PROTEASOMEʼS PROTEOLYTIC ACTIVITIES.
Shock·2003
A NEWLY DISCOVERED NATURAL INHIBITOR OF THE PROTEASOME BLOCKS LPS-INDUCED SHOCK IN ANIMALS.
Shock·2003
THE PROTEASOME MAY BE AN IMPORTANT THERAPEUTIC TARGET IN GRAM-NEGATIVE SEPSIS.
Shock·2003
NOVEL LPS-BINDING PROTEINS IN MURINE MACROPHAGES.
Shock·2002
PROMOTION OF APOPTOSIS BY CHANGES IN NF-κB AND SERUM AMYLOID A ACTIVATING FACTOR AFTER HEMORRHAGE AND RESUSCITATION.
Shock·2002
Natural and synthetic LPS and lipid a analogs or partial structures that antagonize or induce tolerance to LPS.
PubMed·1998· 5 cited
Diphosphoryl lipid A from Rhodobacter sphaeroides inhibits complexes that form in vitro between lipopolysaccharide (LPS)-binding protein, soluble CD14, and spectrally pure LPS
Infection and Immunity·1997· 35 citedOpen Access
Inhibition of lipopolysaccharide-induced transcription factor Sp1 binding by spectrally pure diphosphoryl lipid A from Rhodobacter sphaeroides, protein kinase inhibitor H-8, and dexamethasone
Infection and Immunity·1997· 15 citedOpen Access
Rhodobacter sphaeroides diphosphoryl lipid A inhibits interleukin-6 production in CD14-negative murine marrow stromal ST2 cells stimulated with lipopolysaccharide or paclitaxel (taxol)
Journal of Endotoxin Research·1997· 11 cited
リポ多糖またはパクリタキセル(タキソール)で活性化されたCD14陰性ネズミ髄ストローマST2細胞における、ロドバクタースフェロイデス(rhodobacter sphaeroides)ジホスホリルリピドAによるインターロイキン-6産生の阻害 | 文献情報 | J-GLOBAL 科学技術総合リンクセンター
Journal of Endotoxin Research·1997
リポ多糖またはパクリタキセル(タキソール)で活性化されたCD14陰性ネズミ髄ストローマST2細胞における、ロドバクタースフェロイデス(rhodobacter sphaeroides)ジホスホリルリピドAによるインターロイキン‐6産生の阻害
Journal of Endotoxin Research·1997
Paclitaxel (Taxol)-induced NF-kappaB translocation in murine macrophages
Infection and Immunity·1996· 69 citedOpen Access
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Frequent Co-authors

Koichi Takayama(30), C R Raetz(6), E. Ribi(5), Paolo Mascagni(5), Pin‐Yu Perera(5), Stefanie N. Vogel(5), Robert J. Cotter(4), Douglas T. Golenbock(3), Louise E. Anderson(3), Theo N. Kirkland(3), John L. Cantrell(3), Carl L. Manthey(3), Heinrich K. Schnoes(3), Teruo Kirikae(3), Bruce W. Jarvis(3), Asaf A. Qureshi(2), A. Gapor(2), Y. H. Chong(2), A. S. H. Ong(2), David M. Peterson(2)