Shawn M. Davidson

Northwestern University — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

39h-index10.5kcitations156works6.22yr avg
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Research Topics

Cancer, Hypoxia, and Metabolism(54), Mitochondrial Function and Pathology(28), Prostate Cancer Treatment and Research(20), Metabolism, Diabetes, and Cancer(20), Epigenetics and DNA Methylation(19)

Publications156 total

A metabolic atlas of mouse aging
Cell Metabolism·2025· 4 cited
Figure 3 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
2025Open Access
Figure 2 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
2025Open Access
Figure 4 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
2025Open Access
Figure 1 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
2025Open Access
Figure 5 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
2025Open Access
Figure 7 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
2025Open Access
Figure 6 from Translational Activation of ATF4 through Mitochondrial Anaplerotic Metabolic Pathways Is Required for DLBCL Growth and Survival
2025Open Access
Genetic modulation of mitochondrial NAD+ regeneration does not prevent dopaminergic neuron dysfunction caused by mitochondrial complex I impairment
Frontiers in Cell and Developmental Biology·2025Open Access
Integrated spatial and functional metabolic profiling identified the thick ascending limb as a mitochondrial vulnerability hub in acute kidney injury
Kidney International·2025· 1 citedOpen Access
Genetic modulation of mitochondrial NAD+ regeneration does not prevent dopaminergic neuron dysfunction caused by mitochondrial complex I impairment
2025Open Access
Author Correction: APOE4 impairs myelination via cholesterol dysregulation in oligodendrocytes
Nature·2024· 7 citedOpen Access
Loss of mitochondrial enzyme GPT2 leads to reprogramming of synaptic glutamate metabolism
Molecular Brain·2024· 4 citedOpen Access
Loss of mitochondrial enzyme GPT2 leads to reprogramming of synaptic glutamate metabolism
2024Open Access
Targeting Nucleotides Dependency Via SIRT3 Inhibition to Overcome Metabolic Plasticity and Flexibility of Dlbcls
Blood·2024Open Access
Multi-omic analysis of Huntington’s disease reveals a compensatory astrocyte state
Nature Communications·2024· 26 citedOpen Access
Abstract PO-034: Targeting metabolic plasticity and flexibility via SIRT3 inhibition to restrain nucleotides availability in DLBCLs
Blood Cancer Discovery·2024
Glutamine antagonist DRP-104 suppresses tumor growth and enhances response to checkpoint blockade in <i>KEAP1</i> mutant lung cancer
Science Advances·2024· 52 citedOpen Access
Developmental basis of SHH medulloblastoma heterogeneity
Nature Communications·2024· 24 citedOpen Access
Acidic Methanol Treatment Facilitates Matrix-Assisted Laser Desorption Ionization-Mass Spectrometry Imaging of Energy Metabolism
Analytical Chemistry·2023· 26 citedOpen Access
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Frequent Co-authors

Matthew G. Vander Heiden(25), Joshua D. Rabinowitz(7), Tyler Jacks(7), Thales Papagiannakopoulos(7), Alba Luengo(7), Clary B. Clish(6), Christian M. Metallo(5), Brian P. Fiske(5), Lewis C. Cantley(5), Craig J. Thomas(5), Christopher R. Chin(5), Noel R. Park(5), William J. Israelsen(4), Volkan I. Sayin(4), Sarah E. LeBoeuf(4), Triantafyllia Karakousi(4), Lucas B. Sullivan(4), Elizaveta Freinkman(4), Aaron M. Hosios(4), Tara TeSlaa(4)