Sang-Oh Yoon

Biochemistry, Genetics and Molecular Biology · Life Sciences

25h-index3.8kcitations59works6.32yr avg
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Research Topics

PI3K/AKT/mTOR signaling in cancer(17), Melanoma and MAPK Pathways(11), Cancer, Hypoxia, and Metabolism(7), Protein Kinase Regulation and GTPase Signaling(5), Selenium in Biological Systems(5)

Publications59 total

mTOR inhibition reprograms cellular lipid homeostasis by inducing alternative lipid uptake and promoting cholesterol transport
Molecular Cell·2025· 1 citedOpen Access
Retraction Notice to: The mTORC1/S6K1 Pathway Regulates Glutamine Metabolism through the eIF4B- Dependent Control of c-Myc Translation
Current Biology·2024
The expanding landscape of canonical and non-canonical protein phosphorylation
Trends in Biochemical Sciences·2024· 18 cited
Ran-Binding Protein 3 Phosphorylation Links the Ras and PI3-Kinase Pathways to Nucleocytoplasmic Transport
Molecular Cell·2023· 1 cited
mTOR inhibition reprograms cellular proteostasis by regulating eIF3D-mediated selective mRNA translation and promotes cell phenotype switching
Cell Reports·2023· 25 citedOpen Access
Data from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
2023Open Access
Figure S2 from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
2023Open Access
Data from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
2023Open Access
Figure S2 from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
2023Open Access
Figure S1 from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
2023Open Access
Figure S1 from RSK Regulates PFK-2 Activity to Promote Metabolic Rewiring in Melanoma
2023Open Access
Supporting information_Figure legends from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
Data from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
Figure S3. Elevated levels of reactive oxygen species are responsible for cell death in Tsc2-/- MEFs from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
Figure S4. The combination of BSO and rapamycin induces mitochondrial ROS and alters mitochondrial morphology from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
Figure S3. Elevated levels of reactive oxygen species are responsible for cell death in Tsc2-/- MEFs from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
SI materials and methods from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
SI materials and methods from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
Supporting information_Figure legends from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
Figure S2 Inhibition of mTORC1 and glutamate cysteine ligase induces cytotoxicity in Tsc2-/- MEFs from mTORC1-Driven Tumor Cells Are Highly Sensitive to Therapeutic Targeting by Antagonists of Oxidative Stress
2023Open Access
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Frequent Co-authors

Sejeong Shin(27), John Blenis(19), Philippe P. Roux(12), An‐Sik Chung(8), Chenggang Li(8), Erik Zhang(8), Jane Yu(8), Jianming Zhang(8), Thibault Houlès(6), Min-Joon Han(5), Simon‐Pierre Gravel(5), Geneviève Lavoie(5), Mathilde Savall(5), Antoine Méant(5), Benoı̂t Grondin(5), Louis Gaboury(5), Julie St‐Pierre(5), Yang Sun(5), Steven P. Gygi(4), Andrew Y. Choo(3)