Eileen S. Carpenter

Michigan Medicine — United States

Medicine · Health Sciences

20h-index3.3kcitations276works0.92yr avg
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Research Topics

Pancreatic and Hepatic Oncology Research(137), Immune cells in cancer(89), Phagocytosis and Immune Regulation(55), Epigenetics and DNA Methylation(47), Cancer Immunotherapy and Biomarkers(40)

Publications276 total

Spatial Analysis of Intraductal Papillary Mucinous Neoplasms Defines a Paradoxical Keratin 17-positive, Low-grade Epithelial Population Harboring Malignant Features
Cellular and Molecular Gastroenterology and Hepatology·2026Open Access
Key Resources Table 1 from Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Polarization
2026Open Access
Figure S2 from Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Polarization
2026Open Access
Figure S1 from Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Polarization
2026Open Access
Figure S5 from Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Polarization
2026Open Access
Figure S4 from Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Polarization
2026Open Access
Figure S3 from Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Polarization
2026Open Access
Figure S6 from Aryl Hydrocarbon Receptor Ligands Drive Pancreatic Cancer Initiation and Progression through Protumorigenic T-cell Polarization
2026Open Access
Supplementary Figure S9 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Table 1 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S12 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S8 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S1 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S10 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S7 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S5 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Data from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S6 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S4 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
Supplementary Figure S11 from Fibroblast STAT3 Activation Drives Organ-Specific Premetastatic Niche Formation
2026Open Access
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Frequent Co-authors

Marina Pasca di Magliano(39), Katelyn L. Donahue(29), Filip Bednar(24), Howard C. Crawford(22), Timothy L. Frankel(22), Kristee Brown(22), Zeribe C. Nwosu(21), Nina G. Steele(20), Samantha B. Kemp(19), Carlos E. Espinoza(18), Costas A. Lyssiotis(18), Arvind Rao(18), Padma Kadiyala(17), Stephanie The(15), Rosa E. Menjivar(15), Jiaqi Shi(15), Christopher J. Halbrook(14), Wenting Du(14), Ahmed M. Elhossiny(14), Vaibhav Sahai(13)