Barbara Scott Nelson

University of Michigan — United States

Medicine · Health Sciences

15h-index1.8kcitations44works0.02yr avg
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Research Topics

Pancreatic and Hepatic Oncology Research(14), Cancer, Hypoxia, and Metabolism(12), Epigenetics and DNA Methylation(10), Teacher Education and Leadership Studies(8), Mathematics Education and Teaching Techniques(7)

Publications44 total

Glucose-6-phosphate dehydrogenase deficiency accelerates pancreatic acinar-to-ductal metaplasia
2023· 3 citedOpen Access
87. A tale of two aspirates: sequential pulls of bone marrow yield discordant results in hepatosplenic T-cell lymphoma
Cancer Genetics·2023
Differential integrated stress response and asparagine production drive symbiosis and therapy resistance of pancreatic adenocarcinoma cells
Nature Cancer·2022· 69 citedOpen Access
Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context
eLife·2022· 110 citedOpen Access
Author response: Metabolic requirement for GOT2 in pancreatic cancer depends on environmental context
2022Open Access
Abstract 2176: NADPH production and redox homeostasis regulate acinar to ductal metaplasia
Cancer Research·2022
GOT1 inhibition promotes pancreatic cancer cell death by ferroptosis
Nature Communications·2021· 239 citedOpen Access
DDRE-24. TARGETING PURINE METABOLISM TO OVERCOME GLIOBLASTOMA THERAPY RESISTANCE
Neuro-Oncology Advances·2021· 1 citedOpen Access
The Pancreatic Tumor Microenvironment Compensates for Loss of GOT2
2020· 4 citedOpen Access
Purine metabolism regulates DNA repair and therapy resistance in glioblastoma
Nature Communications·2020· 246 citedOpen Access
Clonal Heterogeneity Supports Mitochondrial Metabolism in Pancreatic Cancer
2020· 8 citedOpen Access
Purine metabolism regulates DNA repair and therapy resistance in glioblastoma
2020· 19 citedOpen Access
GOT1 Inhibition Primes Pancreatic Cancer for Ferroptosis through the Autophagic Release of Labile Iron
2020· 3 citedOpen Access
Tissue of origin dictates GOT1 dependence and confers synthetic lethality to radiotherapy
Cancer & Metabolism·2020· 45 citedOpen Access
MOESM2 of Tissue of origin dictates GOT1 dependence and confers synthetic lethality to radiotherapy
Figshare·2020Open Access
MOESM3 of Tissue of origin dictates GOT1 dependence and confers synthetic lethality to radiotherapy
Figshare·2020Open Access
Defining Roles of Metabolic Reprogramming in Pancreatic Tumorigenesis and Tumor Maintenance
Deep Blue (University of Michigan)·2020Open Access
Tissue of Origin Dictates GOT1 Dependence and Confers Synthetic Lethality to Radiotherapy
2019· 5 citedOpen Access
Abstract 4549: Macrophage-epithelial metabolic crosstalk impairs chemotherapy in pancreatic cancer
Tumor Biology·2019Open Access
Metabolism Drives Carcinogenesis and Maintenance of Pancreatic Tumors
Cancer Discovery·2019· 5 cited
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Frequent Co-authors

Costas A. Lyssiotis(22), Christopher J. Halbrook(20), Howard C. Crawford(14), Peter Sajjakulnukit(12), Li Zhang(10), Marina Pasca di Magliano(10), Daniel M. Kremer(9), Amy L. Myers(9), Zeribe C. Nwosu(9), Johanna Ramos(9), Galloway Thurston(8), Ilya Kovalenko(7), Kari Wilder-Romans(7), Joseph Dresser(7), Mary A. Davis(7), Sriram Chandrasekaran(7), Daniel Wahl(7), Lin Lin(7), Ho‐Joon Lee(6), Yaqing Zhang(6)