Henry W. Long

Brigham and Women's Hospital — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

56h-index12.1kcitations616works1.82yr avg
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Research Topics

Epigenetics and DNA Methylation(82), Cancer Immunotherapy and Biomarkers(79), Monoclonal and Polyclonal Antibodies Research(70), Cancer, Lipids, and Metabolism(64), Immunotherapy and Immune Responses(57)

Publications616 total

Distinct transcription factor interactions drive HOXB13 activity in different stages of prostate cancer
Proceedings of the National Academy of Sciences·2025Open Access
Supplementary Table S9 from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Table S3 from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Table S8 from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Figures from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Table S5 from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Fig. S7 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
Supplementary Fig. S3 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
scATAnno: Automated Cell Type Annotation for Single-cell ATAC Sequencing Data
Genomics Proteomics & Bioinformatics·2025Open Access
Supplementary Table S7 from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Fig. S6 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
Supplementary Fig. S5 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
Supplementary Table S6 from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Table S2 from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Fig. S1 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
Supplementary Fig. S4 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
Supplementary Tables S2-13 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
Supplementary Table S4 from JAK–STAT Signaling in Inflammatory Breast Cancer Enables Chemotherapy-Resistant Cell States
2025Open Access
Supplementary Fig. S8 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
Supplementary Fig. S2 from Endocrine Therapy Synergizes with SMAC Mimetics to Potentiate Antigen Presentation and Tumor Regression in Hormone Receptor–Positive Breast Cancer
2025Open Access
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Frequent Co-authors

Myles Brown(29), Xintao Qiu(20), Paloma Cejas(17), X. Shirley Liu(15), Matthew L. Freedman(15), Prakash K. Rao(13), Klothilda Lim(10), Mark M. Pomerantz(9), Clifford A. Meyer(8), Yingtian Xie(8), Alba Font‐Tello(8), Lewyn Li(8), Leigh Ellis(7), Ji-Heui Seo(7), Kornélia Polyák(6), William C. Hahn(6), Raga Vadhi(6), Massimo Loda(5), Fugen Li(5), Ramesh A. Shivdasani(5)