Arjun Bhutkar

Massachusetts Institute of Technology — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

43h-index12.4kcitations194works29.02yr avg
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Research Topics

Chromatin Remodeling and Cancer(41), Pancreatic and Hepatic Oncology Research(35), Mechanisms of cancer metastasis(29), Pancreatitis Pathology and Treatment(24), Cancer Genomics and Diagnostics(22)

Publications194 total

Intrinsic electrical activity drives small-cell lung cancer progression
Nature·2025· 29 citedOpen Access
1517 Mutant-p53 amplifies Cxcl1 expression from distal enhancers blunting immune checkpoint inhibition efficacy in pancreatic cancer
2023Open Access
21 Increased mutation burden alone is not sufficient to increase immunogenicity of DNA mismatch repair deficient tumors
Regular and Young Investigator Award Abstracts·2023Open Access
Mismatch repair deficiency is not sufficient to elicit tumor immunogenicity
Nature Genetics·2023· 147 citedOpen Access
Tumor-intrinsic LKB1-LIF signaling axis establishes a myeloid niche to promote immune evasion and tumor growth
2023· 4 citedOpen Access
RNA-seq (cell line) Supplementary Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
RNA-seq (cell line) Supplementary Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Bulk ATAC-seq Supplementary Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Supplementary Figures S1-S7 from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Supplementary Table S1 from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
RNA-seq (primary tumors) Supplementary Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Supplementary Figures S1-S7 from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Supplementary Table S1 from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
CUT&RUN Supplementary Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
RNA-seq (primary tumors) Supplementary Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Bulk ATAC-seq Supplementary Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
CUT&RUN Supplementary Data from <i>Smarca4</i> Inactivation Promotes Lineage-Specific Transformation and Early Metastatic Features in the Lung
2023Open Access
Supplementary Figure Legends, Table Legends, Figures S1 - S5 from Stromal Expression of miR-143/145 Promotes Neoangiogenesis in Lung Cancer Development
2023Open Access
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Frequent Co-authors

Tyler Jacks(31), Roderick T. Bronson(12), Francisco J. Sánchez‐Rivera(9), Peter M.K. Westcott(7), Matthew G. Vander Heiden(6), Phillip A. Sharp(6), Thales Papagiannakopoulos(5), Jason M. Schenkel(5), Vasilena Gocheva(5), William M Gelbart(4), Wen Xue(4), Rodrigo Romero(4), Jacqueline A. Lees(4), Talya L. Dayton(4), Kim L. Mercer(4), Aviv Regev(4), Temple F. Smith(4), Mandar D. Muzumdar(4), Kathryn Miller(4), Mary C. Beytagh(4)