Michelle Palmieri

Walter and Eliza Hall Institute of Medical Research — Australia

Medicine · Health Sciences

17h-index1.1kcitations231works0.02yr avg
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Research Topics

Cancer-related Molecular Pathways(160), Cancer Genomics and Diagnostics(21), PARP inhibition in cancer therapy(14), Genetic factors in colorectal cancer(13), Colorectal Cancer Treatments and Studies(12)

Publications231 total

Figure S3 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Figure S1 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Figure S2 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Supplementary Table 1 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Figure S5 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Figure S4 from Genotype-Tailored ERK/MAPK Pathway and HDAC Inhibition Rewires the Apoptotic Rheostat to Trigger Colorectal Cancer Cell Death
2025Open Access
Correction: Combination of bazedoxifene with chemotherapy and SMAC-mimetics for the treatment of colorectal cancer
Cell Death and Disease·2025Open Access
Supplementary Figures from <i>MACROD2</i> Haploinsufficiency Impairs Catalytic Activity of PARP1 and Promotes Chromosome Instability and Growth of Intestinal Tumors
2024Open Access
Combination of bazedoxifene with chemotherapy and SMAC-mimetics for the treatment of colorectal cancer
Cell Death and Disease·2024· 7 citedOpen Access
Data from <i>MACROD2</i> Haploinsufficiency Impairs Catalytic Activity of PARP1 and Promotes Chromosome Instability and Growth of Intestinal Tumors
2024Open Access
Supplementary Data Table S9 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Figure S5 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Figure S7 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Figure S7 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Figure S27 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Table S8 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Figure S27 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Table S5 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Figure S16 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
Supplementary Data Table S6 from Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells
2024Open Access
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Frequent Co-authors

Oliver M. Sieber(41), Peter Gibbs(35), Tao Tan(24), Yumiko Hirokawa(22), Chris Riffkin(19), David C.S. Huang(19), Zilu Wang(18), Matteo Burigotto(18), Sabrina Ghetti(18), François Vaillant(18), Bianca D. Capaldo(18), Lin Tai(18), Daniel S. Simpson(18), Catherine Chang(18), Allan Shuai Huang(18), Elizabeth Lieschke(18), Sarah T. Diepstraten(18), Deeksha Kaloni(18), Connie S.N. Li Wai Suen(18), Alexandra L. Garnham(18)