Carol Jolly

University of Liverpool — United Kingdom

Medicine · Health Sciences

9h-index315citations21works2.32yr avg
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Research Topics

Cancer Immunotherapy and Biomarkers(6), Mitochondrial Function and Pathology(6), Metabolomics and Mass Spectrometry Studies(5), Metabolism and Genetic Disorders(5), Drug-Induced Adverse Reactions(5)

Publications21 total

Mitotoxic effects of the melanin-concentrating hormone receptor 1 (MCHr1) antagonist AZD1979 are influenced by mitochondrial DNA variation in HepG2 cybrids
Toxicology·2025· 1 citedOpen Access
1174 Utility of immune cell receptor repertoires as predictors of development and mechanism of immune checkpoint inhibitor induced pneumonitis
Regular and Young Investigator Award Abstracts·2024Open Access
Greater mechanistic understanding of the cutaneous pathogenesis of Stevens–Johnson syndrome/toxic epidermal necrolysis can shed light on novel therapeutic strategies: a comprehensive review
Current Opinion in Allergy and Clinical Immunology·2024· 6 citedOpen Access
2240P Novel genetic markers associated with immune checkpoint inhibitor induced immune-related adverse events
Annals of Oncology·2023Open Access
The generation of HepG2 transmitochondrial cybrids to reveal the role of mitochondrial genotype in idiosyncratic drug-induced liver injury
eLife·2023· 1 citedOpen Access
<scp>TNF</scp>‐α induced extracellular release of keratinocyte high‐mobility group box 1 in <scp>Stevens‐Johnson</scp> syndrome/toxic epidermal necrolysis: Biomarker and putative mechanism of pathogenesis
The Journal of Dermatology·2023· 9 citedOpen Access
TNF-α‒Mediated Keratinocyte Expression and Release of Matrix Metalloproteinase 9: Putative Mechanism of Pathogenesis in Stevens‒Johnson Syndrome/Toxic Epidermal Necrolysis
Journal of Investigative Dermatology·2022· 21 citedOpen Access
Author response: The generation of HepG2 transmitochondrial cybrids to reveal the role of mitochondrial genotype in idiosyncratic drug-induced liver injury
2022Open Access
The generation of HepG2 transmitochondrial cybrids to reveal the role of mitochondrial genotype in idiosyncratic drug-induced liver injury: a translational <i>in vitro</i> study
2022Open Access
Checkpoint Inhibition Reduces the Threshold for Drug-Specific T-Cell Priming and Increases the Incidence of Sulfasalazine Hypersensitivity
Toxicological Sciences·2021· 34 citedOpen Access
Identification of neutralising pembrolizumab anti-drug antibodies in patients with melanoma
Scientific Reports·2021· 10 citedOpen Access
T cell mediated hypersensitivity to previously tolerated iodinated contrast media precipitated by introduction of atezolizumab
Journal for ImmunoTherapy of Cancer·2021· 23 citedOpen Access
Mucosal-associated invariant T (MAIT) cells are activated in the gastrointestinal tissue of patients with combination ipilimumab and nivolumab therapy-related colitis in a pathology distinct from ulcerative colitis
Clinical & Experimental Immunology·2020· 26 citedOpen Access
The utility of a differentiated preclinical liver model, HepaRG cells, in investigating delayed toxicity via inhibition of mitochondrial-replication induced by fialuridine
Toxicology and Applied Pharmacology·2020· 9 citedOpen Access
Mucosal-associated invariant T (MAIT) cells are activated in the gastrointestinal tissue of patients with combination ipilimumab and nivolumab therapy-related colitis in a pathology distinct from ulcerative colitis.
Authorea·2020· 2 citedOpen Access
Acute Metabolic Switch Assay Using Glucose/Galactose Medium in HepaRG Cells to Detect Mitochondrial Toxicity
Current Protocols in Toxicology·2019· 21 cited
The utility of HepaRG cells for bioenergetic investigation and detection of drug-induced mitochondrial toxicity
Toxicology in Vitro·2018· 42 citedOpen Access
Evaluating Mitotoxicity as Either a Single or Multi‐Mechanistic Insult in the Context of Hepatotoxicity
2018
Transcriptional Repression of E-Cadherin by Human Papillomavirus Type 16 E6
PLoS ONE·2012· 85 citedOpen Access
The effects of leptomycin B on HPV-infected cells
St Andrews Research Repository (St Andrews Research Repository)·2008Open Access
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Frequent Co-authors

Amy E. Chadwick(8), Anna Olsson‐Brown(8), Laleh Kamalian(4), Dominic P. Williams(4), Munir Pirmohamed(4), Benjamin P. Fairfax(4), Daniel F. Carr(4), Amy Ball(4), Oisin Douglas(3), Jan Snoeys(3), Damir Simic(3), Mario Monshouwer(3), B. Kevin Park(3), Sarah C. Sasson(3), Paul Klenerman(3), Oliver Brain(3), Lorenzo Ressel(3), Jonathan J. Lyon(3), Ana Alfirevic(3), Sean Hammond(2)