Eve Kandyba

UCSF Helen Diller Family Comprehensive Cancer Center — United States

Medicine · Health Sciences

14h-index929citations39works1.62yr avg
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Research Topics

Cancer Cells and Metastasis(16), Hair Growth and Disorders(15), Skin and Cellular Biology Research(7), Wound Healing and Treatments(6), Wnt/β-catenin signaling in development and cancer(5)

Publications39 total

0887 Long-lived and genetically-diverse skin organoids derived from murine adult hair follicle stem cells
Journal of Investigative Dermatology·2025Open Access
Data from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 2 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 6 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Table 2 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 5 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 7 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 3 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 1 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Dataset from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 4 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 8 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Table 1 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 9 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
Cancer Discovery·2025· 6 citedOpen Access
Stem-cell states converge in multistage cutaneous squamous cell carcinoma development
Science·2024· 21 cited
The interplay between dormant mutated cells and tumor promotion by chronic tissue damage in determining cancer risk
bioRxiv (Cold Spring Harbor Laboratory)·2024Open Access
Gene networks reveal stem-cell state convergence during preneoplasia and progression to malignancy in multistage skin carcinogenesis
2023· 2 citedOpen Access
Abstract 2198: The impact of carcinogens, obesity, and chronic inflammatory processes on mutational signatures and cancer risk in mouse tumor models
Cancer Research·2022
Multicolour lineage tracing reveals clonal dynamics of squamous carcinoma evolution from initiation to metastasis
Nature Cell Biology·2018· 101 citedOpen Access
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Frequent Co-authors

Allan Balmain(21), Kyle Halliwill(20), Reyno Delrosario(20), Olga K. Mirzoeva(18), Leslie A. Lange(17), Di Wu(16), Quân Tran(16), John DiGiovanni(16), Melissa Quino Reeves(15), Nora Bayani(15), Laura Riva(15), Kavya Achanta(15), Ludmil B. Alexandrov(15), Krzysztof Kobielak(14), S. M. Ashiqul Islam(14), Erik N. Bergstrom(14), Yvonne Leung(4), Yibu Chen(4), Pedro Lee(3), Rupali Gund(3)