Kyle Halliwill

University of California, San Francisco — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

14h-index1.3kcitations74works1.02yr avg
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Research Topics

Cancer Genomics and Diagnostics(21), Single-cell and spatial transcriptomics(21), Cancer Cells and Metastasis(16), Cancer-related molecular mechanisms research(14), Cancer Immunotherapy and Biomarkers(12)

Publications74 total

Pairwise ratio transformation of gene expression data leads to improved checkpoint response prediction in lung cancer patients
BMC Bioinformatics·2025Open Access
Supplementary Methods File S1 from iBRIDGE: A Data Integration Method to Identify Inflamed Tumors from Single-cell RNA-Seq Data and Differentiate Cell Type–Specific Markers of Immune-Cell Infiltration
2025Open Access
Supplementary Figures from iBRIDGE: A Data Integration Method to Identify Inflamed Tumors from Single-cell RNA-Seq Data and Differentiate Cell Type–Specific Markers of Immune-Cell Infiltration
2025Open Access
Supplementary Tables from iBRIDGE: A Data Integration Method to Identify Inflamed Tumors from Single-cell RNA-Seq Data and Differentiate Cell Type–Specific Markers of Immune-Cell Infiltration
2025Open Access
Data from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 2 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 6 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Table 2 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 5 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 7 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 3 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 1 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Dataset from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 4 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 8 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Table 1 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Supplementary Figure 9 from Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
2025Open Access
Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
Cancer Discovery·2025· 6 citedOpen Access
Abstract A099: The IO-Atlas Project: A cross-cohort tumor immunophenotyping platform enhancing precision immuno-oncology
Cancer Immunology Research·2025· 1 cited
Supplementary Figures from iBRIDGE: A Data Integration Method to Identify Inflamed Tumors from Single-Cell RNAseq Data and Differentiate Cell Type–Specific Markers of Immune-Cell Infiltration
2024Open Access
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Frequent Co-authors

Allan Balmain(23), Tolga Turan(19), Josue Samayoa(19), Reyno Delrosario(18), Sarah Kongpachith(17), Eve Kandyba(16), Rebecca Mathew(15), Leslie A. Lange(15), Howard J. Jacob(15), Laura Riva(14), Olga K. Mirzoeva(14), Ludmil B. Alexandrov(13), Di Wu(13), Robert T. McLaughlin(13), Mikhail Binnewies(13), Dhemath Reddy(13), Xi Zhao(13), Shiming Ye(13), Quân Tran(12), Nora Bayani(12)