Zefeng Wang

Central South University — China

Biochemistry, Genetics and Molecular Biology · Life Sciences

50h-index12.9kcitations334works0.72yr avg
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Research Topics

RNA modifications and cancer(125), RNA Research and Splicing(95), RNA and protein synthesis mechanisms(60), Cholangiocarcinoma and Gallbladder Cancer Studies(35), MicroRNA in disease regulation(20)

Publications334 total

Supplementary Table S4 from An Inflammatory Checkpoint Generated by <i>IL1RN</i> Splicing Offers Therapeutic Opportunity for <i>KRAS</i>-Mutant Intrahepatic Cholangiocarcinoma
2025Open Access
Supplementary Table S6 from An Inflammatory Checkpoint Generated by <i>IL1RN</i> Splicing Offers Therapeutic Opportunity for <i>KRAS</i>-Mutant Intrahepatic Cholangiocarcinoma
2025Open Access
Supplementary Table S2 from An Inflammatory Checkpoint Generated by <i>IL1RN</i> Splicing Offers Therapeutic Opportunity for <i>KRAS</i>-Mutant Intrahepatic Cholangiocarcinoma
2025Open Access
Supplementary Table S5 from An Inflammatory Checkpoint Generated by <i>IL1RN</i> Splicing Offers Therapeutic Opportunity for <i>KRAS</i>-Mutant Intrahepatic Cholangiocarcinoma
2025Open Access
Supplementary Table S1 from An Inflammatory Checkpoint Generated by <i>IL1RN</i> Splicing Offers Therapeutic Opportunity for <i>KRAS</i>-Mutant Intrahepatic Cholangiocarcinoma
2025Open Access
Supplementary Table S3 from An Inflammatory Checkpoint Generated by <i>IL1RN</i> Splicing Offers Therapeutic Opportunity for <i>KRAS</i>-Mutant Intrahepatic Cholangiocarcinoma
2025Open Access
Supplementary Table S7 from An Inflammatory Checkpoint Generated by <i>IL1RN</i> Splicing Offers Therapeutic Opportunity for <i>KRAS</i>-Mutant Intrahepatic Cholangiocarcinoma
2025Open Access
Supplementary Figures S1-9 from An Inflammatory Checkpoint Generated by <i>IL1RN</i> Splicing Offers Therapeutic Opportunity for <i>KRAS</i>-Mutant Intrahepatic Cholangiocarcinoma
2025Open Access
Supplementary Table S3 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
2025Open Access
Supplementary Table S1 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
2025Open Access
Supplementary Data from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
2025Open Access
Supplementary Table S5 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
2025Open Access
Supplementary Table S4 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
2025Open Access
Supplementary Table S2 from <i>RBM10</i> Loss Promotes <i>EGFR</i>-Driven Lung Cancer and Confers Sensitivity to Spliceosome Inhibition
2025Open Access
PTEN Loss Promotes PI3Kβ Phosphorylation and EPHA2/SRC/p-PI3KβY962 Complex Assembly to Drive Tumorigenesis
Cancer Discovery·2025· 1 citedOpen Access
A frequency-domain modulation decoupling control method for multibeam precision pointing
2025
Effective in vivo RNA base editing via engineered cytidine deaminase APOBECs fused with PUF proteins
Nature Communications·2025· 1 citedOpen Access
Pan-tissue transcriptome analysis reveals sex-dimorphic human aging
eLife·2025· 1 citedOpen Access
Author response: Pan-tissue transcriptome analysis reveals sex-dimorphic human aging
2025Open Access
A foundation language model to decipher diverse regulation of RNAs
Genome biology·2025· 1 citedOpen Access
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Frequent Co-authors

Yang Wang(12), Xinshu Xiao(7), Christopher B. Burge(6), Xiaojuan Fan(5), Huanhuan Wei(5), James C. Morris(4), Mark E. Drew(4), Paul T. Englund(4), Yi‐Hsuan Tsai(4), Daniel Domínguez(4), Hongguang Jin(4), Catherine C. L. Wong(3), Traci M. Tanaka Hall(3), Rajarshi Choudhury(3), Sirui Zhang(3), Wei Han(3), Yun Yang(2), Miaowei Mao(2), Xiaowei Song(2), Yang Zhang(2)