Valerie P. Sah

University of California, San Diego — United States

Biochemistry, Genetics and Molecular Biology · Life Sciences

16h-index4.3kcitations17works0.02yr avg
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Research Topics

Protein Kinase Regulation and GTPase Signaling(8), Receptor Mechanisms and Signaling(6), Signaling Pathways in Disease(5), Cardiac electrophysiology and arrhythmias(4), Ion channel regulation and function(4)

Publications17 total

Tandem genomic arrangement of a G protein (<i>Gna15</i>) and G protein‐coupled receptor (<i>s1p<sub>4</sub></i>/<i>lp<sub>C1</sub></i>/<i>Edg6</i>) gene
FEBS Letters·2002· 16 citedOpen Access
The Cardiac-specific Nuclear δB Isoform of Ca2+/Calmodulin-dependent Protein Kinase II Induces Hypertrophy and Dilated Cardiomyopathy Associated with Increased Protein Phosphatase 2A Activity
Journal of Biological Chemistry·2002· 241 citedOpen Access
The Rho effector, PKN, regulates ANF gene transcription in cardiomyocytes through a serum response element
American Journal of Physiology-Heart and Circulatory Physiology·2000· 49 cited
The Role of Rho in G Protein-Coupled Receptor Signal Transduction
The Annual Review of Pharmacology and Toxicology·2000· 345 cited
Cardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failure
Journal of Clinical Investigation·1999· 252 citedOpen Access
Enhanced Gαq signaling: A common pathway mediates cardiac hypertrophy and apoptotic heart failure
Proceedings of the National Academy of Sciences·1998· 533 citedOpen Access
Tyrosine Kinase and c-Jun NH<sub>2</sub>-Terminal Kinase Mediate Hypertrophic Responses to Prostaglandin F<sub>2α</sub>in Cultured Neonatal Rat Ventricular Myocytes
Circulation Research·1998· 61 cited
Cardiac hypertrophy induced by mitogen-activated protein kinase kinase 7, a specific activator for c-Jun NH2-terminal kinase in ventricular muscle cells.
Journal of Biological Chemistry·1998· 16 citedOpen Access
Cardiac Hypertrophy Induced by Mitogen-activated Protein Kinase Kinase 7, a Specific Activator for c-Jun NH2-terminal Kinase in Ventricular Muscle Cells
Journal of Biological Chemistry·1998· 338 citedOpen Access
The Low Molecular Weight GTPase Rho Regulates Myofibril Formation and Organization in Neonatal Rat Ventricular Myocytes
Journal of Biological Chemistry·1998· 193 citedOpen Access
Cardiac Muscle Cell Hypertrophy and Apoptosis Induced by Distinct Members of the p38 Mitogen-activated Protein
1998
Cardiac Muscle Cell Hypertrophy and Apoptosis Induced by Distinct Members of the p38 Mitogen-activated Protein Kinase Family
Journal of Biological Chemistry·1998· 853 citedOpen Access
The MEKK-JNK Pathway Is Stimulated by α1-Adrenergic Receptor and Ras Activation and Is Associated with in Vitro and in Vivo Cardiac Hypertrophy
Journal of Biological Chemistry·1997· 235 citedOpen Access
Pathways and roadblocks in muscarinic receptor-mediated growth regulation
Life Sciences·1997· 18 cited
Rho Is Required for Gαq and α1-Adrenergic Receptor Signaling in Cardiomyocytes
Journal of Biological Chemistry·1996· 223 citedOpen Access
Altered Cell Cycle Kinetics, Gene Expression, and G <sub>1</sub> Restriction Point Regulation in <i>Rb</i> -Deficient Fibroblasts
Molecular and Cellular Biology·1996· 304 citedOpen Access
A subset of p53-deficient embryos exhibit exencephaly
Nature Genetics·1995· 580 cited

Frequent Co-authors

Joan Heller Brown(14), Kenneth R. Chien(9), Yibin Wang(6), Jiahuai Han(4), John Ross(3), Shuang Huang(2), Bart O. Williams(2), Tyler Jacks(2), John W. Adams(2), Gerald W. Dorn(2), Michael R. Morissette(2), Masahiko Hoshijima(2), Laura D. Attardi(1), George Mulligan(1), Roderick T. Bronson(1), Yoshihito Sakata(1), Michael Davis(1), Stephen B. Liggett(1), Tammy M. Seasholtz(1), Sarah A. Sagi(1)